SWI/SNF complex heterogeneity is related to polyphenotypic differentiation, prognosis, and immune response in rhabdoid tumors

Abstract Background Rhabdoid tumors (RTs) arise within (atypical teratoid/rhabdoid tumor [AT/RT]) or outside the brain (extra [e]CNS-RT) and are driven mainly by inactivation of the SWItch/sucrose nonfermentable (SWI/SNF) complex subunit SWI/SNF-related matrix-associated actin-dependent regulator of...

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Veröffentlicht in:	Neuro-oncology (Charlottesville, Va.) Va.), 2020-06, Vol.22 (6), p.785-796
Hauptverfasser:	Panwalkar, Pooja, Pratt, Drew, Chung, Chan, Dang, Derek, Le, Paul, Martinez, Daniel, Bayliss, Jill M, Smith, Kyle S, Adam, Mike, Potter, Steven, Northcott, Paul A, Mascarenhas, Leo, Shows, Jared, Pawel, Bruce, Margol, Ashley, Huang, Annie, Judkins, Alexander R, Venneti, Sriram
Format:	Artikel
Sprache:	eng
Schlagworte:	Actins Basic and Translational Investigations Cell Differentiation Child Chromosomal Proteins, Non-Histone - genetics DNA-Binding Proteins - genetics Humans Immunity Prognosis Rhabdoid Tumor - genetics SMARCB1 Protein Sucrose Tumor Microenvironment
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