Prohibitin 2 deficiency impairs cardiac fatty acid oxidation and causes heart failure

Fatty acids are the most major substrate source for adult cardiac energy generation. Prohibitin 2 (PHB2), a highly conserved protein located in mitochondrial inner membrane, plays key roles in cellular energy metabolic homeostasis. However, its functions in regulating cardiac fatty acid metabolism h...

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Veröffentlicht in:	Cell death & disease 2020-03, Vol.11 (3), p.181-181, Article 181
Hauptverfasser:	Wu, Dechao, Jian, Chongshu, Peng, Qi, Hou, Tingting, Wu, Keling, Shang, Bizhi, Zhao, Minglei, Wang, Yanru, Zheng, Wen, Ma, Qi, Li, Chuan-Yun, Cheng, Heping, Wang, Xianhua, Zhao, Ling
Format:	Artikel
Sprache:	eng
Schlagworte:	14 14/19 14/28 14/34 38 38/77 631/80/304 64/110 64/60 692/699/75/230 82/79 Animals Antibodies Biochemistry Biomedical and Life Sciences Cardiomyocytes Carnitine Cell Biology Cell Culture Congestive heart failure Fatty acids Fatty Acids - metabolism Heart failure Heart Failure - metabolism Heart rate Homeostasis Humans Immunology Life Sciences Metabolic disorders Metabolism Mice Mitochondria Oxidation Oxidation-Reduction Prohibitin Repressor Proteins - deficiency
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