The central role of Fas‐ligand cell signaling in inflammatory lung diseases

Following inflammation and injury in the lung, loss of epithelial cell precursors could determine the balance between tissue regeneration and fibrosis. This review discusses evidence that proapoptotic Fas‐Fas ligand (FasL) signaling plays a central role in pulmonary inflammation, injury and fibrosis...

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Veröffentlicht in:	Journal of cellular and molecular medicine 2004-07, Vol.8 (3), p.285-293
Hauptverfasser:	DosReis, G. A., Borges, Valeria M., Zin, W. A.
Format:	Artikel
Sprache:	eng
Schlagworte:	Acute Disease Acute respiratory distress syndrome Alveoli Apoptosis Apoptosis Review Series Cell signaling Chemokines Epithelial cells Epithelial Cells - metabolism Epithelial Cells - pathology Fas ligand Fas Ligand Protein fas Receptor - immunology FasL protein Fibrosis Gene Expression Regulation Humans Inflammation injury Interleukin-1 - metabolism Leukocytes (neutrophilic) lung Lung diseases macrophage Macrophages Macrophages, Alveolar - immunology Macrophages, Alveolar - pathology Membrane Glycoproteins - physiology neutrophil Neutrophils - immunology phagocytosis Pneumonia - immunology Pneumonia - metabolism Pulmonary Alveoli - immunology Pulmonary Alveoli - metabolism Pulmonary Fibrosis - immunology Pulmonary Fibrosis - metabolism Signal Transduction Silicosis Silicosis - immunology
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