Aortic stenosis: Is it simply a degenerative process or an active atherosclerotic process?
Aortic stenosis remains the most common cause of cardiac valve replacement in developed countries. The prevalence of this condition increases with age, and many of the risk factors for coronary artery disease also appear related to the develop‐ment and progression of aortic stenosis. Recent studies...
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| Veröffentlicht in: | Clinical cardiology (Mahwah, N.J.) N.J.), 2005-03, Vol.28 (3), p.111-114 |
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| container_title | Clinical cardiology (Mahwah, N.J.) |
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| creator | Hughes, Bradley R. Chahoud, Georges Mehta, J. L. |
| description | Aortic stenosis remains the most common cause of cardiac valve replacement in developed countries. The prevalence of this condition increases with age, and many of the risk factors for coronary artery disease also appear related to the develop‐ment and progression of aortic stenosis. Recent studies also suggest a relationship between calcium and lipid accumulation in both coronary artery disease and aortic stenosis. Most important, there is growing evidence that aggressive treatment of coronary artery disease risk factors, particularly hyperlipi‐demia, may influence the progression of aortic stenosis. In this article the current literature is reviewed as it relates to vascular biology, pathogenesis of aortic valve disease, and current and newly emerging management approaches in the care of the patient with aortic stenosis. |
| doi_str_mv | 10.1002/clc.4960280303 |
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L.</creatorcontrib><title>Aortic stenosis: Is it simply a degenerative process or an active atherosclerotic process?</title><title>Clinical cardiology (Mahwah, N.J.)</title><addtitle>Clin Cardiol</addtitle><description>Aortic stenosis remains the most common cause of cardiac valve replacement in developed countries. The prevalence of this condition increases with age, and many of the risk factors for coronary artery disease also appear related to the develop‐ment and progression of aortic stenosis. Recent studies also suggest a relationship between calcium and lipid accumulation in both coronary artery disease and aortic stenosis. Most important, there is growing evidence that aggressive treatment of coronary artery disease risk factors, particularly hyperlipi‐demia, may influence the progression of aortic stenosis. 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Vascular system</subject><subject>Coronary Artery Disease - pathology</subject><subject>Disease Progression</subject><subject>Editor's Note</subject><subject>Endocardial and cardiac valvular diseases</subject><subject>Heart</subject><subject>Humans</subject><subject>Medical sciences</subject><subject>statins</subject><subject>valvular heart disease</subject><issn>0160-9289</issn><issn>1932-8737</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2005</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNqFkc1vFCEYh4nR2LV69Wi4aE-z8gLDhwdNs_GjySZe9OKFMAzTYthhhdma_e_LuhO3XuwFCDw8vLw_hF4CWQIh9K2Lbsm1IFQRRtgjtADNaKMkk4_RgoAgjaZKn6FnpfysPFGUPUVn0CpgAtoF-nGZ8hQcLpMfUwnlHb4qOEy4hM027rHFvb_2o892Crceb3NyvhScMrYjtu7Ppp1ufE7FxToeVDP04Tl6MthY_It5PkffP338tvrSrL9-vlpdrhvXMmBNP4CDDrqOsk5Y2muvLeWk7fq6HHrKqe40B68oB8qY6jWnxHIGtutBasXO0fujd7vrNr53fpyyjWabw8bmvUk2mH9PxnBjrtOtEaLljNMquJgFOf3a-TKZTSjOx2hHn3bFSM4FEC1FJd_8lxRSktr7h0EKFBSXsoLLI-hqC0v2w9-6gZhDwqYmbE4J1wuv7v_2hM-RVuD1DNjibByyHV0oJ04cVOrA6SP3O0S_f-BZs1qv7hVxB81GwAs</recordid><startdate>200503</startdate><enddate>200503</enddate><creator>Hughes, Bradley R.</creator><creator>Chahoud, Georges</creator><creator>Mehta, J. 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| subjects | aortic stenosis Aortic Valve Stenosis - pathology Aortic Valve Stenosis - physiopathology Aortic Valve Stenosis - therapy atherosclerosis Atherosclerosis (general aspects, experimental research) Biological and medical sciences Blood and lymphatic vessels Cardiology. Vascular system Coronary Artery Disease - pathology Disease Progression Editor's Note Endocardial and cardiac valvular diseases Heart Humans Medical sciences statins valvular heart disease |
| title | Aortic stenosis: Is it simply a degenerative process or an active atherosclerotic process? |
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