Expression and Molecular Regulation of the Cox2 Gene in Gastroenteropancreatic Neuroendocrine Tumors and Antiproliferation of Nonsteroidal Anti-Inflammatory Drugs (NSAIDs)

BACKGROUND Gastroenteropancreatic neuroendocrine tumors (GEP-NETs) has had a significant increase over the past 4 decades. The pathophysiological role of the cyclooxygenase-2 (cox-2) gene and factors responsible for the expression in GEP-NETs is of clinical value. Current study determined the expres...

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Veröffentlicht in:	Medical science monitor 2018-11, Vol.24, p.8125-8140
Hauptverfasser:	Gao, Feng, Zafar, Mohammad Ishraq, Jüttner, Stefan, Höcker, Michael, Wiedenmann, Bertram
Format:	Artikel
Sprache:	eng
Schlagworte:	Anti-Inflammatory Agents, Non-Steroidal - pharmacology Apoptosis - drug effects Cell Cycle - drug effects Cell Line, Tumor Cell Proliferation - drug effects Cyclooxygenase 2 - biosynthesis Cyclooxygenase 2 - genetics Cyclooxygenase Inhibitors - pharmacology Gene Expression Regulation, Enzymologic Gene Expression Regulation, Neoplastic Humans Immunohistochemistry Intestinal Neoplasms - drug therapy Intestinal Neoplasms - enzymology Intestinal Neoplasms - genetics Intestinal Neoplasms - pathology Lab/In Vitro Research Neuroendocrine Tumors - drug therapy Neuroendocrine Tumors - enzymology Neuroendocrine Tumors - genetics Neuroendocrine Tumors - pathology Pancreatic Neoplasms - drug therapy Pancreatic Neoplasms - enzymology Pancreatic Neoplasms - genetics Pancreatic Neoplasms - pathology Stomach Neoplasms - drug therapy Stomach Neoplasms - enzymology Stomach Neoplasms - genetics Stomach Neoplasms - pathology
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