Zinc regulates vascular endothelial cell activity through zinc-sensing receptor ZnR/GPR39

Zn is an essential element for cell survival/growth, and its deficiency is linked to many disorders. Extracellular Zn concentration changes participate in modulating fundamental cellular processes such as proliferation, secretion, ion transport, and cell signal transduction in a mechanism that is no...

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Veröffentlicht in:	American Journal of Physiology: Cell Physiology 2018-04, Vol.314 (4), p.C404-C414
Hauptverfasser:	Zhu, Donghui, Su, Yingchao, Zheng, Yufeng, Fu, Bingmei, Tang, Liping, Qin, Yi-Xian
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Calcium Signaling - drug effects Capillary Permeability - drug effects Cell Adhesion - drug effects Cell Movement - drug effects Cell Proliferation - drug effects Cell Survival - drug effects Cells, Cultured Chlorides - metabolism Chlorides - pharmacology Endothelial Cells - drug effects Endothelial Cells - metabolism Humans Mice, Knockout Neovascularization, Physiologic - drug effects Phosphatidylinositol 3-Kinase - metabolism Receptors, G-Protein-Coupled - drug effects Receptors, G-Protein-Coupled - genetics Receptors, G-Protein-Coupled - metabolism Zinc Compounds - metabolism Zinc Compounds - pharmacology
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