Self‐administration of methamphetamine alters gut biomarkers of toxicity

Methamphetamine (METH) is a highly abused psychostimulant that is associated with an increased risk for developing Parkinson's disease (PD). This enhanced vulnerability likely relates to the toxic effects of METH that overlap with PD pathology, for example, aberrant functioning of α‐synuclein a...

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Veröffentlicht in:	The European journal of neuroscience 2017-08, Vol.46 (3), p.1918-1932
Hauptverfasser:	Flack, Amanda, Persons, Amanda L., Kousik, Sharanya M., Celeste Napier, T., Moszczynska, Anna
Format:	Artikel
Sprache:	eng
Schlagworte:	Biomarkers catecholamines Central nervous system Colon Degeneration Dopamine Dopamine β-monooxygenase Enteric nervous system Gastrointestinal tract Hydroxylase Methamphetamine Motor task performance Movement disorders Myenteric plexus Nervous system Neurodegenerative diseases Neurons Neurotoxicity Operant conditioning parkin Parkinson's disease rat Rats Rodents Self-administration Synuclein Toxicity Tyrosine 3-monooxygenase α‐synuclein
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