Renal Fanconi Syndrome and Hypophosphatemic Rickets in the Absence of Xenotropic and Polytropic Retroviral Receptor in the Nephron

Tight control of extracellular and intracellular inorganic phosphate (Pi) levels is critical to most biochemical and physiologic processes. Urinary Pi is freely filtered at the kidney glomerulus and is reabsorbed in the renal tubule by the action of the apical sodium-dependent phosphate transporters...

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Veröffentlicht in:	Journal of the American Society of Nephrology 2017-04, Vol.28 (4), p.1073-1078
Hauptverfasser:	Ansermet, Camille, Moor, Matthias B, Centeno, Gabriel, Auberson, Muriel, Hu, Dorothy Zhang, Baron, Roland, Nikolaeva, Svetlana, Haenzi, Barbara, Katanaeva, Natalya, Gautschi, Ivan, Katanaev, Vladimir, Rotman, Samuel, Koesters, Robert, Schild, Laurent, Pradervand, Sylvain, Bonny, Olivier, Firsov, Dmitri
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Brief Communications Fanconi Syndrome - etiology Female Male Mice Nephrons Receptors, G-Protein-Coupled - physiology Receptors, Virus - physiology Rickets, Hypophosphatemic - etiology
Online-Zugang:	Volltext
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