The quantal catecholamine release from mouse chromaffin cells challenged with repeated ACh pulses is regulated by the mitochondrial Na+/Ca2+ exchanger

Key points Upon repeated application of short ACh pulses to C57BL6J mouse chromaffin cells, the amperometrically monitored secretory responses promptly decayed to a steady‐state level of around 25% of the initial response. A subsequent K+ pulse, however, overcame such decay. These data suggest that...

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Veröffentlicht in:	The Journal of physiology 2017-03, Vol.595 (6), p.2129-2146
Hauptverfasser:	López‐Gil, Angela, Nanclares, Carmen, Méndez‐López, Iago, Martínez‐Ramírez, Carmen, los Rios, Cristóbal, Padín‐Nogueira, J. Fernando, Montero, Mayte, Gandía, Luis, García, Antonio G.
Format:	Artikel
Sprache:	eng
Schlagworte:	acetylcholine C57BL6J Cellular and Molecular Neuroscience GCP37157 ITH12662 mitochondrial sodium/calcium exchanger mouse chromaffin cells Neuroscience Neuroscience ‐ Cellular/Molecular quantal release of catecholamine Research Paper
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