Absence of deficits in social behaviors and ultrasonic vocalizations in later generations of mice lacking neuroligin4

Mutations in NLGN4X have been identified in individuals with autism spectrum disorders and other neurodevelopmental disorders. A previous study reported that adult male mice lacking neuroligin4 (Nlgn4) displayed social approach deficits in the three‐chambered test, altered aggressive behaviors and r...

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Veröffentlicht in:	Genes, brain and behavior brain and behavior, 2012-11, Vol.11 (8), p.928-941
Hauptverfasser:	Ey, E., Yang, M., Katz, A. M., Woldeyohannes, L., Silverman, J. L., Leblond, C. S., Faure, P., Torquet, N., Le Sourd, A.‐M., Bourgeron, T., Crawley, J. N.
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult social interaction Animals Autism Autism Spectrum Disorder Autism Spectrum Disorder - genetics Behavior Behavior, Animal Cell Adhesion Molecules, Neuronal Cell Adhesion Molecules, Neuronal - genetics Disease Models, Animal Female Genotype juvenile social interaction Life Sciences Male Mice mouse models Mutation Neurobiology neuroligin4 Neurons and Cognition Phenotype resident–intruder Rodents Social Behavior three‐chambered social approach task ultrasonic vocalizations Ultrasonics Vocalization, Animal
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