VEGF164 isoform specific regulation of T‐cell‐dependent experimental colitis in mice

Background: Inflammatory bowel disease (IBD) consists of Crohn's disease (CD) and ulcerative colitis (UC), two widespread diseases of unknown, multifactorial etiology. Colitis pathology involves a pathological angiogenic response where increases in vascular density participate in colitis histop...

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Veröffentlicht in:	Inflammatory bowel diseases 2011-07, Vol.17 (7), p.1501-1512
Hauptverfasser:	Chidlow, John H., Glawe, John D., Pattillo, Christopher B., Pardue, Sibile, Zhang, Songlin, Kevil, Christopher G.
Format:	Artikel
Sprache:	eng
Schlagworte:	Angiogenesis Animal models Animals Aptamers Aptamers, Nucleotide - pharmacology CD4-Positive T-Lymphocytes - immunology colitis Colitis - etiology Colitis - metabolism Colitis - pathology Colon Colon - immunology Colon - metabolism Colon - pathology Crohn's disease Disease Models, Animal Enzyme-Linked Immunosorbent Assay Etiology Homeodomain Proteins Humans IBD inflammation Inflammation - prevention & control Inflammatory bowel diseases Intestine Leukocyte Common Antigens - physiology Lymphocytes T Mice Mice, Inbred C57BL Mice, Transgenic Neovascularization, Pathologic - prevention & control Protein Isoforms T cell T-Lymphocytes, Regulatory - immunology Ulcerative colitis Vascular endothelial growth factor Vascular Endothelial Growth Factor A - antagonists & inhibitors Vascular Endothelial Growth Factor A - genetics Vascular Endothelial Growth Factor A - physiology VEGF
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