The crossroads of inflammation, fibrosis, and arrhythmia following myocardial infarction

Abstract Optimal healing of damaged tissue following myocardial infarction (MI) requires a coordinated cellular response that can be divided into three phases: inflammatory, proliferative/reparative, and maturation. The inflammatory phase, characterized by rapid influx of cytokines, chemokines, and...

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Veröffentlicht in:	Journal of molecular and cellular cardiology 2016-02, Vol.91, p.114-122
Hauptverfasser:	Francis Stuart, Samantha D, De Jesus, Nicole M, Lindsey, Merry L, Ripplinger, Crystal M
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Arrhythmia Arrhythmias, Cardiac - complications Arrhythmias, Cardiac - genetics Arrhythmias, Cardiac - metabolism Arrhythmias, Cardiac - pathology Calcium - metabolism Cardiovascular Fibrosis Gene Expression Regulation Humans Inflammation Interleukin-1beta - genetics Interleukin-1beta - metabolism Interleukin-6 - genetics Interleukin-6 - metabolism Ion Transport Matrix Metalloproteinases - genetics Matrix Metalloproteinases - metabolism Myocardial infarction Myocardial Infarction - complications Myocardial Infarction - genetics Myocardial Infarction - metabolism Myocardial Infarction - pathology Myocardium - metabolism Myocardium - pathology Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism Signal Transduction Tumor Necrosis Factor-alpha - genetics Tumor Necrosis Factor-alpha - metabolism
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