RAP1-GTPase signaling and platelet function

Platelets are critical for hemostasis, i.e., the body’s ability to prevent blood loss at sites of vascular injury. They patrol the vasculature in a quiescent, non-adhesive state for approximately 10 days, after which they are removed from circulation by phagocytic cells of the reticulo-endothelial s...

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Veröffentlicht in:	Journal of molecular medicine (Berlin, Germany) Germany), 2016-01, Vol.94 (1), p.13-19
Hauptverfasser:	Stefanini, Lucia, Bergmeier, Wolfgang
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Biomedical and Life Sciences Biomedicine Blood Platelets - physiology Calcium - metabolism Guanine Nucleotide Exchange Factors - antagonists & inhibitors Guanine Nucleotide Exchange Factors - metabolism Human Genetics Humans Internal Medicine Mice Mice, Knockout Molecular Medicine Phosphatidylinositol 4,5-Diphosphate - metabolism Platelet Activation - physiology Platelet Adhesiveness - physiology rap1 GTP-Binding Proteins - antagonists & inhibitors rap1 GTP-Binding Proteins - metabolism Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors Receptors, Cytoplasmic and Nuclear - genetics Receptors, Cytoplasmic and Nuclear - metabolism Receptors, Purinergic P2Y12 - metabolism Review Signal Transduction Telomere-Binding Proteins - antagonists & inhibitors Telomere-Binding Proteins - metabolism Vascular System Injuries - physiopathology
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