Neuronal copper homeostasis susceptibility by genetic defects in dysbindin, a schizophrenia susceptibility factor

Environmental factors and susceptible genomes interact to determine the risk of neurodevelopmental disorders. Although few genes and environmental factors have been linked, the intervening cellular and molecular mechanisms connecting a disorder susceptibility gene with environmental factors remain m...

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Veröffentlicht in:	Human molecular genetics 2015-10, Vol.24 (19), p.5512-5523
Hauptverfasser:	Gokhale, Avanti, Vrailas-Mortimer, Alysia, Larimore, Jennifer, Comstra, Heather S, Zlatic, Stephanie A, Werner, Erica, Manvich, Daniel F, Iuvone, P Michael, Weinshenker, David, Faundez, Victor
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenosine Triphosphatases - genetics Adenosine Triphosphatases - metabolism Animals Cation Transport Proteins - genetics Cation Transport Proteins - metabolism Cells, Cultured Copper - metabolism Copper-Transporting ATPases Disease Models, Animal Drosophila Drosophila - metabolism Drosophila Proteins - genetics Drosophila Proteins - metabolism Dysbindin Dystrophin-Associated Proteins - genetics Dystrophin-Associated Proteins - metabolism Genetic Predisposition to Disease Hippocampus - metabolism Mice Neurons - metabolism Original Schizophrenia - genetics
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