Angiotensin receptors alter myocardial infarction-induced remodeling of the guinea pig cardiac plexus

Neurohumoral remodeling is fundamental to the evolution of heart disease. This study examined the effects of chronic treatment with an ACE inhibitor (captopril, 3 mg·kg(-1)·day(-1)), AT1 receptor antagonist (losartan, 3 mg·kg(-1)·day(-1)), or AT2 receptor agonist (CGP42112A, 0.14 mg·kg(-1)·day(-1))...

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Veröffentlicht in:	American journal of physiology. Regulatory, integrative and comparative physiology integrative and comparative physiology, 2015-07, Vol.309 (2), p.R179-R188
Hauptverfasser:	Hardwick, Jean C, Ryan, Shannon E, Powers, Emily N, Southerland, E Marie, Ardell, Jeffrey L
Format:	Artikel
Sprache:	eng
Schlagworte:	ACE inhibitors Action Potentials Angiotensin II - pharmacology Angiotensin II Type 1 Receptor Blockers - pharmacology Angiotensin II Type 2 Receptor Blockers - pharmacology Animals Cardiovascular and Renal Integration Disease Models, Animal Electric Stimulation Evoked Potentials Guinea Pigs Heart - innervation Heart attacks Male Myocardial Infarction - metabolism Myocardial Infarction - pathology Myocardial Infarction - physiopathology Neural Control Neurons Norepinephrine - pharmacology Presynaptic Terminals - drug effects Presynaptic Terminals - metabolism Receptor, Angiotensin, Type 1 - drug effects Receptor, Angiotensin, Type 1 - metabolism Receptor, Angiotensin, Type 2 - drug effects Receptor, Angiotensin, Type 2 - metabolism Receptors, Presynaptic - antagonists & inhibitors Receptors, Presynaptic - metabolism Rodents Signal Transduction Time Factors
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