Endothelial Dysfunction, Arterial Stiffening, and Intima-Media Thickening in Large Arteries from HIV-1 Transgenic Mice

HIV patients on highly active antiretroviral therapy (HAART) exhibit elevated incidence of cardiovascular disease (CVD), including a higher risk of myocardial infarction and prevalence of atherosclerotic lesions, as well as increases in markers of subclinical atherosclerosis including increased caro...

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Veröffentlicht in:	Annals of biomedical engineering 2013-04, Vol.41 (4), p.682-693
Hauptverfasser:	Hansen, Laura, Parker, Ivana, Sutliff, Roy L., Platt, Manu O., Gleason, Rudolph L.
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Antiretroviral agents Atherosclerosis - etiology Atherosclerosis - pathology Atherosclerosis - physiopathology Biochemistry Biological and Medical Physics Biomechanics Biomedical and Life Sciences Biomedical Engineering Biomedical Engineering and Bioengineering Biomedicine Biophysics Cardiovascular diseases Cardiovascular Diseases - etiology Cardiovascular Diseases - pathology Cardiovascular Diseases - physiopathology Classical Mechanics Endothelium, Vascular - physiopathology Endothelium, Vascular - virology Genes, Viral HIV Infections - complications HIV-1 - genetics HIV-1 - pathogenicity Human immunodeficiency virus 1 Human Immunodeficiency Virus Proteins - genetics Human Immunodeficiency Virus Proteins - physiology Humans Male Mice Mice, Transgenic Myocardial infarction Proteins Risk factors Tunica Intima - pathology Tunica Intima - virology Tunica Media - pathology Tunica Media - virology Vascular Stiffness - physiology
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