Hypernatremia secondary to post-stroke hypodipsia: just add water
Disorders in water metabolism may occur in stroke patients. When hypernatremia arises in this setting, it is usually secondary to the development of central diabetes insipidus or it is the result of neurologic lesions that prevent patients from having free access to water. Much rarer are the cases o...
Gespeichert in:
Veröffentlicht in: | Clinical kidney journal 2011-08, Vol.4 (4), p.236-237 |
---|---|
Hauptverfasser: | , , |
Format: | Artikel |
Sprache: | eng |
Schlagworte: | |
Online-Zugang: | Volltext |
Tags: |
Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!
|
container_end_page | 237 |
---|---|
container_issue | 4 |
container_start_page | 236 |
container_title | Clinical kidney journal |
container_volume | 4 |
creator | Ramthun, Maikel Mocelin, Altair Jacob Alvares Delfino, Vinicius Daher |
description | Disorders in water metabolism may occur in stroke patients. When hypernatremia arises in this setting, it is usually secondary to the development of central diabetes insipidus or it is the result of neurologic lesions that prevent patients from having free access to water. Much rarer are the cases of post-stroke hypernatremia caused by hypodipsia secondary to lesions of the thirst center. We report the case of a patient with severe hypernatremia, probably secondary to post-hemorrhagic stroke hypodipsia. The hypernatremia seen in this case was corrected by scheduling the patient’s water intake. |
doi_str_mv | 10.1093/ndtplus/sfr057 |
format | Article |
fullrecord | <record><control><sourceid>oup_pubme</sourceid><recordid>TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4421453</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><oup_id>10.1093/ndtplus/sfr057</oup_id><sourcerecordid>10.1093/ndtplus/sfr057</sourcerecordid><originalsourceid>FETCH-LOGICAL-c391t-78dbb6470fd04056490702c61819f00653ab8b88f5900bc2549bf2ded62432433</originalsourceid><addsrcrecordid>eNqFUM9LwzAUDqK4MXf1KL166PbSJm3iQRhDnTDwoueQNqnr3JqQpEr_ezs6h558PHgPvh_v8SF0jWGGgafzRgW7a_3cVw5ofobGCRAWM4rT89MOdISm3m-hrx4BQi_RKKGccMLYGC1WndWukcHpfS0jr0vTKOm6KJjIGh9iH5z50NGms0bV1tfyLtq2PkRSqehLBu2u0EUld15Pj3OC3h4fXpereP3y9LxcrOMy5TjEOVNFkZEcKgUEaEY45JCUGWaYVwAZTWXBCsYqygGKMqGEF1WitMoSkvadTtD94GvbYq9VqZvg5E5YV-_7f4WRtfiLNPVGvJtPQUiCCT0YzAaD0hnvna5OWgzikKc45imGPHvBze-LJ_pPej3hdiCY1v5n9g2rr4M9</addsrcrecordid><sourcetype>Open Access Repository</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype></control><display><type>article</type><title>Hypernatremia secondary to post-stroke hypodipsia: just add water</title><source>Oxford Journals Open Access Collection</source><source>Elektronische Zeitschriftenbibliothek - Frei zugängliche E-Journals</source><source>PubMed Central</source><creator>Ramthun, Maikel ; Mocelin, Altair Jacob ; Alvares Delfino, Vinicius Daher</creator><creatorcontrib>Ramthun, Maikel ; Mocelin, Altair Jacob ; Alvares Delfino, Vinicius Daher</creatorcontrib><description>Disorders in water metabolism may occur in stroke patients. When hypernatremia arises in this setting, it is usually secondary to the development of central diabetes insipidus or it is the result of neurologic lesions that prevent patients from having free access to water. Much rarer are the cases of post-stroke hypernatremia caused by hypodipsia secondary to lesions of the thirst center. We report the case of a patient with severe hypernatremia, probably secondary to post-hemorrhagic stroke hypodipsia. The hypernatremia seen in this case was corrected by scheduling the patient’s water intake.</description><identifier>ISSN: 2048-8505</identifier><identifier>ISSN: 1753-0784</identifier><identifier>EISSN: 2048-8513</identifier><identifier>EISSN: 1753-0792</identifier><identifier>DOI: 10.1093/ndtplus/sfr057</identifier><identifier>PMID: 25949488</identifier><language>eng</language><publisher>England: Oxford University Press</publisher><subject>II. Clinical Reports</subject><ispartof>Clinical kidney journal, 2011-08, Vol.4 (4), p.236-237</ispartof><rights>The Author 2011. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com 2011</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c391t-78dbb6470fd04056490702c61819f00653ab8b88f5900bc2549bf2ded62432433</citedby><cites>FETCH-LOGICAL-c391t-78dbb6470fd04056490702c61819f00653ab8b88f5900bc2549bf2ded62432433</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>230,314,776,780,881,1598,27901,27902</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/25949488$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Ramthun, Maikel</creatorcontrib><creatorcontrib>Mocelin, Altair Jacob</creatorcontrib><creatorcontrib>Alvares Delfino, Vinicius Daher</creatorcontrib><title>Hypernatremia secondary to post-stroke hypodipsia: just add water</title><title>Clinical kidney journal</title><addtitle>NDT Plus</addtitle><description>Disorders in water metabolism may occur in stroke patients. When hypernatremia arises in this setting, it is usually secondary to the development of central diabetes insipidus or it is the result of neurologic lesions that prevent patients from having free access to water. Much rarer are the cases of post-stroke hypernatremia caused by hypodipsia secondary to lesions of the thirst center. We report the case of a patient with severe hypernatremia, probably secondary to post-hemorrhagic stroke hypodipsia. The hypernatremia seen in this case was corrected by scheduling the patient’s water intake.</description><subject>II. Clinical Reports</subject><issn>2048-8505</issn><issn>1753-0784</issn><issn>2048-8513</issn><issn>1753-0792</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2011</creationdate><recordtype>article</recordtype><sourceid>TOX</sourceid><recordid>eNqFUM9LwzAUDqK4MXf1KL166PbSJm3iQRhDnTDwoueQNqnr3JqQpEr_ezs6h558PHgPvh_v8SF0jWGGgafzRgW7a_3cVw5ofobGCRAWM4rT89MOdISm3m-hrx4BQi_RKKGccMLYGC1WndWukcHpfS0jr0vTKOm6KJjIGh9iH5z50NGms0bV1tfyLtq2PkRSqehLBu2u0EUld15Pj3OC3h4fXpereP3y9LxcrOMy5TjEOVNFkZEcKgUEaEY45JCUGWaYVwAZTWXBCsYqygGKMqGEF1WitMoSkvadTtD94GvbYq9VqZvg5E5YV-_7f4WRtfiLNPVGvJtPQUiCCT0YzAaD0hnvna5OWgzikKc45imGPHvBze-LJ_pPej3hdiCY1v5n9g2rr4M9</recordid><startdate>20110801</startdate><enddate>20110801</enddate><creator>Ramthun, Maikel</creator><creator>Mocelin, Altair Jacob</creator><creator>Alvares Delfino, Vinicius Daher</creator><general>Oxford University Press</general><scope>TOX</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>5PM</scope></search><sort><creationdate>20110801</creationdate><title>Hypernatremia secondary to post-stroke hypodipsia: just add water</title><author>Ramthun, Maikel ; Mocelin, Altair Jacob ; Alvares Delfino, Vinicius Daher</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c391t-78dbb6470fd04056490702c61819f00653ab8b88f5900bc2549bf2ded62432433</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2011</creationdate><topic>II. Clinical Reports</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Ramthun, Maikel</creatorcontrib><creatorcontrib>Mocelin, Altair Jacob</creatorcontrib><creatorcontrib>Alvares Delfino, Vinicius Daher</creatorcontrib><collection>Oxford Journals Open Access Collection</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>PubMed Central (Full Participant titles)</collection><jtitle>Clinical kidney journal</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Ramthun, Maikel</au><au>Mocelin, Altair Jacob</au><au>Alvares Delfino, Vinicius Daher</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Hypernatremia secondary to post-stroke hypodipsia: just add water</atitle><jtitle>Clinical kidney journal</jtitle><addtitle>NDT Plus</addtitle><date>2011-08-01</date><risdate>2011</risdate><volume>4</volume><issue>4</issue><spage>236</spage><epage>237</epage><pages>236-237</pages><issn>2048-8505</issn><issn>1753-0784</issn><eissn>2048-8513</eissn><eissn>1753-0792</eissn><abstract>Disorders in water metabolism may occur in stroke patients. When hypernatremia arises in this setting, it is usually secondary to the development of central diabetes insipidus or it is the result of neurologic lesions that prevent patients from having free access to water. Much rarer are the cases of post-stroke hypernatremia caused by hypodipsia secondary to lesions of the thirst center. We report the case of a patient with severe hypernatremia, probably secondary to post-hemorrhagic stroke hypodipsia. The hypernatremia seen in this case was corrected by scheduling the patient’s water intake.</abstract><cop>England</cop><pub>Oxford University Press</pub><pmid>25949488</pmid><doi>10.1093/ndtplus/sfr057</doi><tpages>2</tpages><oa>free_for_read</oa></addata></record> |
fulltext | fulltext |
identifier | ISSN: 2048-8505 |
ispartof | Clinical kidney journal, 2011-08, Vol.4 (4), p.236-237 |
issn | 2048-8505 1753-0784 2048-8513 1753-0792 |
language | eng |
recordid | cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_4421453 |
source | Oxford Journals Open Access Collection; Elektronische Zeitschriftenbibliothek - Frei zugängliche E-Journals; PubMed Central |
subjects | II. Clinical Reports |
title | Hypernatremia secondary to post-stroke hypodipsia: just add water |
url | https://sfx.bib-bvb.de/sfx_tum?ctx_ver=Z39.88-2004&ctx_enc=info:ofi/enc:UTF-8&ctx_tim=2025-02-19T12%3A37%3A41IST&url_ver=Z39.88-2004&url_ctx_fmt=infofi/fmt:kev:mtx:ctx&rfr_id=info:sid/primo.exlibrisgroup.com:primo3-Article-oup_pubme&rft_val_fmt=info:ofi/fmt:kev:mtx:journal&rft.genre=article&rft.atitle=Hypernatremia%20secondary%20to%20post-stroke%20hypodipsia:%20just%20add%20water&rft.jtitle=Clinical%20kidney%20journal&rft.au=Ramthun,%20Maikel&rft.date=2011-08-01&rft.volume=4&rft.issue=4&rft.spage=236&rft.epage=237&rft.pages=236-237&rft.issn=2048-8505&rft.eissn=2048-8513&rft_id=info:doi/10.1093/ndtplus/sfr057&rft_dat=%3Coup_pubme%3E10.1093/ndtplus/sfr057%3C/oup_pubme%3E%3Curl%3E%3C/url%3E&disable_directlink=true&sfx.directlink=off&sfx.report_link=0&rft_id=info:oai/&rft_id=info:pmid/25949488&rft_oup_id=10.1093/ndtplus/sfr057&rfr_iscdi=true |