Cortical parvalbumin and somatostatin GABA neurons express distinct endogenous modulators of nicotinic acetylcholine receptors

Inhibition from GABAergic interneurons in brain circuits is a critical component of cognitive function. This inhibition is regulated through a diverse network of neuromodulation. A number of recent studies suggest that one of the major regulators of interneuron function is nicotinic acetylcholinergi...

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Veröffentlicht in:	Molecular brain 2014-10, Vol.7 (1), p.75-75, Article 75
Hauptverfasser:	Demars, Michael P, Morishita, Hirofumi
Format:	Artikel
Sprache:	eng
Schlagworte:	Analysis Animals Brain Cognitive ability Experiments GABAergic Neurons - metabolism Glutamic Acid - metabolism Health aspects Hybridization Interneurons Labeling Lynx Medicine Membrane Glycoproteins - metabolism Mental illness Mice, Inbred C57BL Nerve Tissue Proteins - metabolism Neurons Neuropeptides - metabolism Parvalbumins - metabolism Receptors, Nicotinic - metabolism Receptors, Serotonin, 5-HT3 - metabolism Rodents Schizophrenia Somatostatin - metabolism Visual Cortex - metabolism
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