Smoking accelerates aging of the small airway epithelium

Aging involves multiple biologically complex processes characterized by a decline in cellular homeostasis over time leading to a loss and impairment of physiological integrity and function. Specific cellular hallmarks of aging include abnormal gene expression patterns, shortened telomeres and associ...

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Veröffentlicht in:	Respiratory research 2014-09, Vol.15 (1), p.94-94, Article 94
Hauptverfasser:	Walters, Matthew S, De, Bishnu P, Salit, Jacqueline, Buro-Auriemma, Lauren J, Wilson, Timothy, Rogalski, Allison M, Lief, Lindsay, Hackett, Neil R, Staudt, Michelle R, Tilley, Ann E, Harvey, Ben-Gary, Kaner, Robert J, Mezey, Jason G, Ashbridge, Beth, Moore, Malcolm A S, Crystal, Ronald G
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Age Aging Analysis Biotechnology industry Carbon monoxide Case-Control Studies Cellular Senescence - drug effects Cellular Senescence - genetics Cigarettes Data analysis Epithelial Cells - chemistry Epithelial Cells - drug effects Epithelial Cells - pathology Female Gene expression Gene Expression Profiling Gene Expression Regulation Genes Health aspects Homeostasis Humans Lung diseases Male Middle Aged Nicotine Physiological aspects Physiology Population Respiratory function Respiratory Mucosa - chemistry Respiratory Mucosa - drug effects Respiratory Mucosa - pathology RNA, Messenger - analysis Smoke - adverse effects Smoking Smoking - adverse effects Smoking - genetics Smoking - pathology Standard deviation Statistical analysis Studies Telomerase Telomere - genetics Telomere Shortening Telomeres Toxins Urine
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