Interleukin-33-Dependent Innate Lymphoid Cells Mediate Hepatic Fibrosis

Liver fibrosis is a consequence of chronic liver diseases and thus a major cause of mortality and morbidity. Clinical evidence and animal studies suggest that local tissue homeostasis is disturbed due to immunological responses to chronic hepatocellular stress. Poorly defined stress-associated infla...

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Veröffentlicht in:	Immunity (Cambridge, Mass.) Mass.), 2013-08, Vol.39 (2), p.357-371
Hauptverfasser:	Mchedlidze, Tamar, Waldner, Maximilian, Zopf, Steffen, Walker, Jennifer, Rankin, Andrew L., Schuchmann, Marcus, Voehringer, David, McKenzie, Andrew N.J., Neurath, Markus F., Pflanz, Stefan, Wirtz, Stefan
Format:	Artikel
Sprache:	eng
Schlagworte:	Adoptive Transfer Animals Cell Proliferation Cells, Cultured Chemokines Confidence intervals Hepatic Stellate Cells - metabolism Immune system Inflammation Interleukin-13 - metabolism Interleukin-33 Interleukins - immunology Interleukins - metabolism Liver - cytology Liver - immunology Liver - metabolism Liver cirrhosis Liver Cirrhosis - immunology Lymphocyte Activation Lymphocytes - immunology Lymphocytes - metabolism Mice Mice, Inbred BALB C Mice, Inbred C57BL Mice, Knockout Mortality Protein expression Proteins R&D Receptors, Interleukin-4, Type II - metabolism Research & development Rodents Signal Transduction STAT6 Transcription Factor - metabolism Viral infections
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