Voltage-Gated Sodium Channel Expression in Mouse DRG after SNI Leads to Re-Evaluation of Projections of Injured Fibers

Background: Dysregulation of voltage-gated sodium channels (Navs) is believed to play a major role in nerve fiber hyperexcitability associated with neuropathic pain. A complete transcriptional characterization of the different isoforms of Navs under normal and pathological conditions had never been...

Ausführliche Beschreibung

Gespeichert in:

Bibliographische Detailangaben
Veröffentlicht in:	Molecular pain 2014-03, Vol.10 (1), p.19-19
Hauptverfasser:	Laedermann, Cédric J, Pertin, Marie, Suter, Marc R, Decosterd, Isabelle
Format:	Artikel
Sprache:	eng
Schlagworte:	Activating transcription factor 3 Activating Transcription Factor 3 - metabolism Analysis Analysis of Variance Anesthesiology Animal models Animals Channel gating Colleges & universities Disease Models, Animal Dorsal root ganglia Down-regulation ELAV Proteins - metabolism Fibers Ganglia, Spinal - metabolism Ganglia, Spinal - pathology Gene Expression Regulation - physiology Genetic aspects Genetic transcription Injuries Isoforms Localization Lumbar Vertebrae Mice Mice, Inbred C57BL Neuralgia Neuralgia - metabolism Neuralgia - pathology Neurons Neurons, Afferent - metabolism Neurons, Afferent - pathology Pain Physiological aspects Proteins Rodents Sciatic Nerve - pathology Sodium channels (voltage-gated) Software Spinal nerves Spinal Nerves - injuries Sural nerve Voltage-Gated Sodium Channels - genetics Voltage-Gated Sodium Channels - metabolism
Online-Zugang:	Volltext
Tags:	Tag hinzufügen Keine Tags, Fügen Sie den ersten Tag hinzu!

Schreiben Sie den ersten Kommentar!