N -Methyl-D-Aspartate Receptor and Calbindin-Containing Neurons in the Anterior Cingulate Cortex in Schizophrenia and Bipolar Disorder

Background Glutamatergic modulation of γ-aminobutyric acid (GABA) interneurons via the NR2A subunit of the N -methyl-D-aspartate (NMDA) receptor in the cerebral cortex contributes to the pathophysiology of schizophrenia and bipolar disorder. Previously, we found that, in the anterior cingulate corte...

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Veröffentlicht in:	Biological psychiatry (1969) 2008-11, Vol.64 (9), p.803-809
Hauptverfasser:	Woo, Tsung-Ung W, Shrestha, Kevin, Lamb, Dorian, Minns, Martin M, Benes, Francine M
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Aged Aged, 80 and over Analysis of Variance Autoradiography Bipolar disorder Bipolar Disorder - pathology Calbindins Case-Control Studies Cohort Studies Female GABA Gene Expression - physiology Gyrus Cinguli - metabolism Gyrus Cinguli - pathology Humans Male Middle Aged Neurons - metabolism Postmortem Changes postmortem human brain Psychiatry Receptors, N-Methyl-D-Aspartate - genetics Receptors, N-Methyl-D-Aspartate - metabolism S100 Calcium Binding Protein G - metabolism schizophrenia Schizophrenia - pathology Schizophrenia - physiopathology
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