Prenatal ethanol exposure disrupts intraneocortical circuitry, cortical gene expression, and behavior in a mouse model of FASD

In utero ethanol exposure from a mother's consumption of alcoholic beverages impacts brain and cognitive development, creating a range of deficits in the child (Levitt, 1998; Lebel et al., 2012). Children diagnosed with fetal alcohol spectrum disorders (FASD) are often born with facial dysmorph...

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Veröffentlicht in:	The Journal of neuroscience 2013-11, Vol.33 (48), p.18893-18905
Hauptverfasser:	El Shawa, Hani, Abbott, 3rd, Charles W, Huffman, Kelly J
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Behavior, Animal - drug effects Cadherins - biosynthesis Cadherins - genetics Cell Count Cerebral Cortex - drug effects Cerebral Cortex - metabolism Cerebral Cortex - pathology Ethanol - blood Female Fetal Alcohol Spectrum Disorders - genetics Fetal Alcohol Spectrum Disorders - physiopathology Fetal Alcohol Spectrum Disorders - psychology Gene Expression - drug effects In Vitro Techniques Inhibitor of Differentiation Protein 2 - biosynthesis Inhibitor of Differentiation Protein 2 - genetics Mice Microscopy, Fluorescence Nuclear Receptor Subfamily 1, Group F, Member 2 - biosynthesis Nuclear Receptor Subfamily 1, Group F, Member 2 - genetics Osmolar Concentration Pregnancy Pregnancy, Animal - drug effects Prenatal Exposure Delayed Effects - genetics Prenatal Exposure Delayed Effects - physiopathology Prenatal Exposure Delayed Effects - psychology Weight Gain
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