Activation of mitochondrial function and Hb expression in non‐haematopoietic cells by an EPO inducer ameliorates ischaemic diseases in mice

Background and Purpose Many organs suffer from ischaemic injuries that reduce their ability to generate sufficient energy, which is required for functional maintenance and repair. Erythropoietin (EPO) ameliorates ischaemic injuries by pleiotropic effects. The aim of this study was to investigate the...

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Veröffentlicht in:	British journal of pharmacology 2013-08, Vol.169 (7), p.1461-1476
Hauptverfasser:	Hsu, Pei‐Lun, Horng, Lin‐Yea, Peng, Kang‐Yung, Wu, Chia‐Ling, Sung, Hui‐Ching, Wu, Rong‐Tsun
Format:	Artikel
Sprache:	eng
Schlagworte:	anaemia Animals Biosynthesis cardiomyopathy Cardiovascular Diseases - drug therapy Cardiovascular Diseases - metabolism Cell Line Cells, Cultured Cisplatin - pharmacology Doxorubicin - pharmacology endurance performance erythropoietin Erythropoietin - agonists Erythropoietin - metabolism Glucosides - pharmacology haemoglobin HEK293 Cells Hemoglobins - metabolism Hepatocytes - metabolism Humans inducer ischaemic disease Kidney - metabolism Male Medical research Mice Mice, Inbred C57BL mitochondria Mitochondria - drug effects Mitochondria - physiology Myocytes, Cardiac - metabolism non‐haematopoietic cell Receptors, Erythropoietin - agonists Research Papers Rodents Stilbenes - pharmacology
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