The role of plasminogen activator inhibitor-1 in gastric mucosal protection

Gastric mucosal health is maintained in response to potentially damaging luminal factors. Aspirin and nonsteroidal anti-inflammatory drugs (NSAIDs) disrupt protective mechanisms leading to bleeding and ulceration. The plasminogen activator system has been implicated in fibrinolysis following gastric...

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Veröffentlicht in:	American journal of physiology: Gastrointestinal and liver physiology 2013-05, Vol.304 (9), p.G814-G822
Hauptverfasser:	Kenny, Susan, Steele, Islay, Lyons, Suzanne, Moore, Andrew R, Murugesan, Senthil V, Tiszlavicz, Laszlo, Dimaline, Rod, Pritchard, D Mark, Varro, Andrea, Dockray, Graham J
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Aspirin - pharmacology Calcium Cells Dinoprostone Ethanol - toxicity Female Gastric Mucosa - drug effects Gene expression Humans Immunohistochemistry Indomethacin - toxicity Male Mice Mucosal Biology Plasminogen Activator Inhibitor 1 - biosynthesis Plasminogen Activator Inhibitor 1 - physiology RNA, Messenger - metabolism Rodents Stomach Urokinase-Type Plasminogen Activator - biosynthesis
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