Follistatin and Follistatin Like-3 Differentially Regulate Adiposity and Glucose Homeostasis

Transforming growth factor‐β superfamily ligands, including activin and myostatin, modulate body composition, islet function, and glucose homeostasis. Their bioactivity is controlled by the antagonists follistatin (FST) and FST like‐3 (FSTL3). The hypothesis tested was that FST and FSTL3 have distin...

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Veröffentlicht in:	Obesity (Silver Spring, Md.) Md.), 2011-10, Vol.19 (10), p.1940-1949
Hauptverfasser:	Brown, Melissa L., Bonomi, Lara, Ungerleider, Nathan, Zina, Jessica, Kimura, Fuminori, Mukherjee, Abir, Sidis, Yisrael, Schneyer, Alan
Format:	Artikel
Sprache:	eng
Schlagworte:	Adipose Tissue - metabolism Animals Blood Glucose - metabolism Body Composition - genetics Body Composition - physiology Body fat Body Fluid Compartments - metabolism Body Weight - physiology Fasting Follistatin - genetics Follistatin - metabolism Follistatin-Related Proteins - genetics Follistatin-Related Proteins - metabolism Genotype Glucose Glucose Intolerance - genetics Glucose Intolerance - metabolism Homeostasis Insulin - metabolism Insulin Resistance - genetics Insulin Resistance - physiology Islets of Langerhans - physiology Male Mice Mice, Inbred C57BL Mice, Knockout Mutation Obesity - genetics Obesity - metabolism Pancreas - metabolism Protein Isoforms Proteins Rodents
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