α1-Adrenergic Receptors Positively Regulate Toll-Like Receptor Cytokine Production from Human Monocytes and Macrophages

Catecholamines released from the sympathetic nervous system in response to stress or injury affect expression of inflammatory cytokines generated by immune cells. α1-Adrenergic receptors (ARs) are expressed on innate immune cell populations, but their subtype expression patterns and signaling charac...

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Veröffentlicht in:	The Journal of pharmacology and experimental therapeutics 2011-08, Vol.338 (2), p.648-657
Hauptverfasser:	Grisanti, Laurel A., Woster, Andrew P., Dahlman, Julie, Sauter, Edward R., Combs, Colin K., Porter, James E.
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Cell Differentiation - immunology Cell Line, Transformed Cells, Cultured Humans Immunity, Innate Inflammation Mediators - physiology Inflammation, Immunopharmacology, and Asthma Interleukin-1beta - biosynthesis Interleukin-1beta - blood Lipopolysaccharides - physiology Macrophages - immunology Macrophages - metabolism Macrophages - pathology Monocytes - immunology Monocytes - metabolism Monocytes - pathology Protein Subunits - biosynthesis Protein Subunits - blood Protein Subunits - physiology Receptors, Adrenergic, alpha-1 - biosynthesis Receptors, Adrenergic, alpha-1 - blood Receptors, Adrenergic, alpha-1 - physiology Signal Transduction - immunology Toll-Like Receptor 4 - biosynthesis Toll-Like Receptor 4 - blood Toll-Like Receptor 4 - physiology Up-Regulation - immunology
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