Early Innate Recognition of Herpes Simplex Virus in Human Primary Macrophages Is Mediated via the MDA5/MAVS-Dependent and MDA5/MAVS/RNA Polymerase III-Independent Pathways

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Veröffentlicht in:Journal of Virology 2010-11, Vol.84 (21), p.11350-11358
Hauptverfasser: Melchjorsen, Jesper, Rintahaka, Johanna, Søby, Stine, Horan, Kristy A, Poltajainen, Alina, Østergaard, Lars, Paludan, Søren R, Matikainen, Sampsa
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container_end_page 11358
container_issue 21
container_start_page 11350
container_title Journal of Virology
container_volume 84
creator Melchjorsen, Jesper
Rintahaka, Johanna
Søby, Stine
Horan, Kristy A
Poltajainen, Alina
Østergaard, Lars
Paludan, Søren R
Matikainen, Sampsa
description Article Usage Stats Services JVI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit StumbleUpon Twitter current issue Spotlights in the Current Issue JVI About JVI Subscribers Authors Reviewers Advertisers Inquiries from the Press Permissions & Commercial Reprints ASM Journals Public Access Policy JVI RSS Feeds 1752 N Street N.W. • Washington DC 20036 202.737.3600 • 202.942.9355 fax • journals@asmusa.org Print ISSN: 0022-538X Online ISSN: 1098-5514 Copyright © 2014 by the American Society for Microbiology.   For an alternate route to JVI .asm.org, visit: JVI       
doi_str_mv 10.1128/JVI.01106-10
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subjects Adaptor Proteins, Signal Transducing - metabolism
Biological and medical sciences
Cells, Cultured
Cytokines - biosynthesis
DEAD Box Protein 58
DEAD-box RNA Helicases - immunology
DEAD-box RNA Helicases - metabolism
Fundamental and applied biological sciences. Psychology
Herpes simplex virus
Humans
Immunity, Innate
Interferon-Induced Helicase, IFIH1
Interferons - biosynthesis
Macrophages - virology
Microbiology
Miscellaneous
Receptors, Immunologic
RNA Polymerase III - metabolism
Signal Transduction - immunology
Simplexvirus - immunology
Virology
Virus-Cell Interactions
title Early Innate Recognition of Herpes Simplex Virus in Human Primary Macrophages Is Mediated via the MDA5/MAVS-Dependent and MDA5/MAVS/RNA Polymerase III-Independent Pathways
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