Comparative epigenomics of human and mouse mammary tumors

Gene silencing by aberrant epigenetic chromatin alteration is a well‐recognized event contributing to tumorigenesis. Although genetically engineered tumor‐prone mouse models have proven a powerful tool in understanding many aspects of carcinogenesis, to date few studies have focused on epigenetic al...

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Veröffentlicht in:	Genes chromosomes & cancer 2009-01, Vol.48 (1), p.83-97
Hauptverfasser:	Demircan, Berna, Dyer, Lisa M., Gerace, Mallory, Lobenhofer, Edward K., Robertson, Keith D., Brown, Kevin D.
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Azacitidine - analogs & derivatives Azacitidine - pharmacology Breast Neoplasms - genetics Breast Neoplasms - metabolism Cell Cycle Proteins - metabolism Cell Line, Tumor Databases, Genetic DNA Methylation DNA Modification Methylases - antagonists & inhibitors DNA Modification Methylases - metabolism Epigenesis, Genetic Gene Expression Regulation Gene Silencing Genes, Tumor Suppressor Glycoproteins - metabolism Histone Deacetylase Inhibitors Histone Deacetylases - metabolism Histone Deacetylases - pharmacology Humans Hydroxamic Acids - pharmacology Mammary Neoplasms, Animal - genetics Mammary Neoplasms, Animal - metabolism Mice Oligonucleotide Array Sequence Analysis Polymerase Chain Reaction Structure-Activity Relationship Tissue Inhibitor of Metalloproteinase-3 - metabolism
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