The Lipoprivic Control of Feeding Is Governed by Fat Metabolism, Not by Leptin or Adipose Depletion

A lipoprivic control of feeding has been proposed based on the finding that appetite is stimulated by drugs such as β-mercaptoacetate (MA) that reduce fatty acid oxidation. The adipose-derived hormone, leptin, has effects on feeding and fat oxidation that are opposite those produced by MA. However,...

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Veröffentlicht in:	Endocrinology (Philadelphia) 2010-05, Vol.151 (5), p.2087-2096
Hauptverfasser:	Hudson, Bryan D, Emanuel, Alan J, Wiater, Michael F, Ritter, Sue
Format:	Artikel
Sprache:	eng
Schlagworte:	Adipose tissue Adipose Tissue - drug effects Adipose Tissue - metabolism Animals Biological and medical sciences Body fat Body weight Body Weight - drug effects Capsaicin Capsaicin - pharmacology Depletion Dietary Fats - administration & dosage Drug metabolism Eating - drug effects Fat metabolism Fats - metabolism Feeding Feeding Behavior - drug effects Fundamental and applied biological sciences. Psychology Leptin Leptin - blood Leptin - metabolism Leptin - pharmacology Lipid Metabolism - drug effects Male Oxidation Rats Rats, Sprague-Dawley Sensory System Agents - pharmacology Thioglycolates - pharmacology Triglycerides - blood Vertebrates: endocrinology
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