Dopamine-Stimulated Dephosphorylation of Connexin 36 Mediates AII Amacrine Cell Uncoupling

Gap junction proteins form the substrate for electrical coupling between neurons. These electrical synapses are widespread in the CNS and serve a variety of important functions. In the retina, connexin 36 (Cx36) gap junctions couple AII amacrine cells and are a requisite component of the high-sensit...

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Veröffentlicht in:	The Journal of neuroscience 2009-11, Vol.29 (47), p.14903-14911
Hauptverfasser:	Kothmann, W. Wade, Massey, Stephen C, O'Brien, John
Format:	Artikel
Sprache:	eng
Schlagworte:	Amacrine Cells - cytology Amacrine Cells - drug effects Amacrine Cells - metabolism Animals Cell Communication - drug effects Cell Communication - physiology Connexins - metabolism Cyclic AMP-Dependent Protein Kinases - drug effects Cyclic AMP-Dependent Protein Kinases - metabolism Dopamine - metabolism Dopamine - pharmacology Enzyme Activation - drug effects Enzyme Activation - physiology Gap Junction delta-2 Protein Gap Junctions - drug effects Gap Junctions - metabolism Gap Junctions - ultrastructure Organ Culture Techniques Phosphorylation - drug effects Protein Phosphatase 1 - metabolism Protein Phosphatase 2 - drug effects Protein Phosphatase 2 - metabolism Rabbits Retina - cytology Retina - drug effects Retina - metabolism Signal Transduction - drug effects Signal Transduction - physiology Synaptic Transmission - physiology Vision, Ocular - drug effects Vision, Ocular - physiology
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