Effects of acute and repeated exposure to lipopolysaccharide on cytokine and corticosterone production during remyelination

Abstract Chronic exposure to the copper-chelating agent, cuprizone (CPZ), is an increasingly popular model for producing demyelination. More importantly, cessation of cuprizone exposure allows for full remyelination, which represents a window of opportunity for determining the influence of environme...

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Veröffentlicht in:	Brain, behavior, and immunity behavior, and immunity, 2007-10, Vol.21 (7), p.962-974
Hauptverfasser:	Urbach-Ross, Daniella, Kusnecov, Alexander W
Format:	Artikel
Sprache:	eng
Schlagworte:	Acute Disease Allergy and Immunology Animals Body Weight CD11b Antigen - metabolism Chelating Agents - toxicity Chronic Disease Corpus Callosum - immunology Corpus Callosum - pathology Corticosterone - blood Cuprizone Cuprizone - toxicity Cytokines Cytokines - metabolism Demyelinating Diseases - chemically induced Demyelinating Diseases - immunology Demyelinating Diseases - pathology Demyelination Disease Models, Animal Leukocyte Common Antigens - metabolism Lipopolysaccharide Lipopolysaccharides - pharmacology Male Mice Mice, Inbred C57BL Microglia Microglia - immunology Myelin Basic Protein - metabolism Myelin Sheath - immunology Myelin Sheath - metabolism Myelin Sheath - pathology Nerve Regeneration - drug effects Nerve Regeneration - immunology Psychiatry Remyelination Spleen - metabolism
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