Interactions between NO, CO and an endothelium‐derived hyperpolarizing factor (EDHF) in maintaining patency of the ductus arteriosus in the mouse

Background and purpose: Prenatal patency of ductus arteriosus is maintained by prostaglandin (PG) E2, possibly along with nitric oxide (NO) and carbon monoxide (CO), and cyclooxygenase (COX) deletion upregulates NO. Here, we have examined enzyme source and action of NO for ductus patency and whether...

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Veröffentlicht in:	British journal of pharmacology 2007-05, Vol.151 (1), p.54-62
Hauptverfasser:	Baragatti, B, Brizzi, F, Barogi, S, Laubach, V E, Sodini, D, Shesely, E G, Regan, R F, Coceani, F
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Biological and medical sciences Biological Factors - physiology bradykinin Bradykinin - pharmacology carbon monoxide Carbon Monoxide - physiology Cardiology. Vascular system Congenital heart diseases. Malformations of the aorta, pulmonary vessels and vena cava cyclooxygenase ductus arteriosus Ductus Arteriosus, Patent - etiology endothelium‐derived hyperpolarizing factor fetal and neonatal physiology Heart heme oxygenase Heme Oxygenase (Decyclizing) - physiology Medical sciences Mice Mice, Inbred C57BL nitric oxide Nitric Oxide - physiology nitric oxide synthase Nitric Oxide Synthase Type II - genetics Nitric Oxide Synthase Type III - physiology Pharmacology. Drug treatments Research Papers
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