Soluble complement receptor one (sCR1) inhibits the development and progression of rat collagen‐induced arthritis

We set out to determine whether inhibition of complement using sCR1 could influence the development and progression of collagen arthritis in the Lewis rat. Collagen arthritis was successfully established in the Lewis rat, using a novel immunization schedule. In separate experiments, cobra venom fact...

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Veröffentlicht in:	Clinical and experimental immunology 2000-01, Vol.119 (1), p.210-216
Hauptverfasser:	Goodfellow, R. M., Williams, A. S., Levin, J. L., Williams, B. D., Morgan, B. P.
Format:	Artikel
Sprache:	eng
Schlagworte:	animal models Animals Arthritis - drug therapy Arthritis - etiology Arthritis - prevention & control Arthritis, Rheumatoid - drug therapy Arthritis, Rheumatoid - etiology Arthritis, Rheumatoid - prevention & control Biological and medical sciences Collagen - immunology Complement Inactivator Proteins - therapeutic use complement inhibition Disease Models, Animal Elapid Venoms - therapeutic use Immunopathology Immunotherapy (general aspects) Male Medical sciences Rats Rats, Inbred Lew Receptors, Complement 3b - therapeutic use Rheumatic Disease sCR1 Solubility Time Factors
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