Cytokine mRNA expression in intestinal tissue of interleukin-2 deficient mice with bowel inflammation

Background—Mice deficient in interleukin-2 (IL-2) develop inflammatory bowel disease resembling ulcerative colitis in humans. Recent studies provided evidence that αβ T cells, particularly CD4 T cells, rather than B cells, are involved in the pathogenesis of bowel inflammation of IL-2 deficient mice...

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Veröffentlicht in:	Gut 1997-12, Vol.41 (6), p.793-800
Hauptverfasser:	Autenrieth, I B, Bucheler, N, Bohn, E, Heinze, G, Horak, I
Format:	Artikel
Sprache:	eng
Schlagworte:	Actins - genetics Animals Biological and medical sciences bowel inflammation Colon Colon - immunology cytokine Cytokines - genetics Cytokines - metabolism Gastroenterology. Liver. Pancreas. Abdomen Gene Expression Inflammation Inflammatory bowel disease Inflammatory Bowel Diseases - immunology Inflammatory Bowel Diseases - metabolism Interferon-gamma - genetics Interleukin-1 - genetics Interleukin-10 - genetics Interleukin-2 - deficiency interleukin-2 deficient mice Interleukin-6 - genetics Intestinal Mucosa - metabolism Intestine, Small - immunology Intestines - immunology Lymphocytes Medical sciences Mice Mice, Inbred C57BL Mice, Mutant Strains mRNA expression Other diseases. Semiology Polymerase Chain Reaction RNA, Messenger - analysis RNA, Messenger - metabolism Stomach. Duodenum. Small intestine. Colon. Rectum. Anus Studies T cell receptors Transforming Growth Factor beta - genetics Tumor Necrosis Factor-alpha - genetics
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