Gene expression in temporal lobe epilepsy is consistent with increased release of glutamate by astrocytes

Patients with temporal lobe epilepsy (TLE) often have a shrunken hippocampus that is known to be the location in which seizures originate. The role of the sclerotic hippocampus in the causation and maintenance of seizures in temporal lobe epilepsy (TLE) has remained incompletely understood despite e...

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Veröffentlicht in:	Molecular medicine (Cambridge, Mass.) Mass.), 2007-01, Vol.13 (1-2), p.1-13
Hauptverfasser:	Lee, Tih-Shih, Mane, Shrikant, Eid, Tore, Zhao, Hongyu, Lin, Aiping, Guan, Zhong, Kim, Jung H, Schweitzer, Jeffrey, King-Stevens, David, Weber, Peter, Spencer, Susan S, Spencer, Dennis D, de Lanerolle, Nihal C
Format:	Artikel
Sprache:	eng
Schlagworte:	Adolescent Adult Age of Onset Astrocytes - metabolism Child Child, Preschool Cytoskeletal Proteins - metabolism DNA - genetics Epilepsy, Temporal Lobe - genetics Epilepsy, Temporal Lobe - metabolism Female Gene Expression Gene Expression Profiling Glial Fibrillary Acidic Protein - metabolism Glutamic Acid - metabolism Hippocampus - metabolism Hippocampus - pathology Hippocampus - surgery Humans Immunohistochemistry Male Membrane Proteins - metabolism Microfilament Proteins - metabolism Middle Aged Models, Biological Nerve Growth Factors - metabolism Oligonucleotide Array Sequence Analysis Phosphoproteins - metabolism Plectin - metabolism Reproducibility of Results Reverse Transcriptase Polymerase Chain Reaction S100 Calcium Binding Protein beta Subunit S100 Proteins - metabolism Transcription, Genetic
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