Fusion of the ALK Gene to the Clathrin Heavy Chain Gene, CLTC, in Inflammatory Myofibroblastic Tumor
Inflammatory myofibroblastic tumor (IMT) is a rare, but distinctive mesenchymal neoplasm composed of fascicles of bland myofibroblasts admixed with a prominent inflammatory component. Genetic studies of IMTs have demonstrated chromosomal abnormalities of 2p23 and rearrangement of the anaplastic lymp...
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| Veröffentlicht in: | The American journal of pathology 2001-08, Vol.159 (2), p.411-415 |
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| creator | Bridge, Julia A. Kanamori, Masahiko Ma, Zhigui Pickering, Diane Hill, D. Ashley Lydiatt, William Lui, Man Yee Colleoni, Gisele W.B. Antonescu, Cristina R. Ladanyi, Marc Morris, Stephan W. |
| description | Inflammatory myofibroblastic tumor (IMT) is a rare, but distinctive mesenchymal neoplasm composed of fascicles of bland myofibroblasts admixed with a prominent inflammatory component. Genetic studies of IMTs have demonstrated chromosomal abnormalities of 2p23 and rearrangement of the anaplastic lymphoma kinase (
ALK) gene locus. In a subset of IMTs, the ALK C-terminal kinase domain is fused with a tropomyosin N-terminal coiled-coil domain. In the current study, fusion of
ALK with the clathrin heavy chain (
CTLC) gene localized to 17q23 was detected in two cases of IMT. One of these cases exhibited a 2;17 translocation in addition to other karyotypic anomalies [46,XX,t(2;17)(p23;q23),add(16)(q24)]. |
| doi_str_mv | 10.1016/S0002-9440(10)61711-7 |
| format | Article |
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ALK) gene locus. In a subset of IMTs, the ALK C-terminal kinase domain is fused with a tropomyosin N-terminal coiled-coil domain. In the current study, fusion of
ALK with the clathrin heavy chain (
CTLC) gene localized to 17q23 was detected in two cases of IMT. One of these cases exhibited a 2;17 translocation in addition to other karyotypic anomalies [46,XX,t(2;17)(p23;q23),add(16)(q24)].</description><identifier>ISSN: 0002-9440</identifier><identifier>EISSN: 1525-2191</identifier><identifier>DOI: 10.1016/S0002-9440(10)61711-7</identifier><identifier>PMID: 11485898</identifier><identifier>CODEN: AJPAA4</identifier><language>eng</language><publisher>Bethesda, MD: Elsevier Inc</publisher><subject>Adult ; Amino Acid Sequence ; Base Sequence ; Biological and medical sciences ; Child, Preschool ; Chromosome Mapping ; Chromosomes, Human, Pair 2 ; Clathrin - genetics ; Dermatology ; Female ; Gene Rearrangement ; Granuloma, Plasma Cell - genetics ; Granuloma, Plasma Cell - pathology ; Head and Neck Neoplasms - genetics ; Head and Neck Neoplasms - pathology ; Humans ; In Situ Hybridization, Fluorescence ; Karyotyping ; Male ; Medical sciences ; Oncogene Proteins, Fusion - genetics ; Pelvic Neoplasms - genetics ; Pelvic Neoplasms - pathology ; Protein-Tyrosine Kinases - genetics ; Receptor Protein-Tyrosine Kinases - genetics ; Reverse Transcriptase Polymerase Chain Reaction ; Short Communications ; Tumors of the skin and soft tissue. Premalignant lesions</subject><ispartof>The American journal of pathology, 2001-08, Vol.159 (2), p.411-415</ispartof><rights>2001 American Society for Investigative Pathology</rights><rights>2002 INIST-CNRS</rights><rights>Copyright American Society for Investigative Pathology Aug 2001</rights><rights>Copyright © 2001, American Society for Investigative Pathology</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c617t-d9f5070e0b2cef011090d03eedd2a5b81c56d315f6fde4370ba85f2cc98c3c463</citedby><cites>FETCH-LOGICAL-c617t-d9f5070e0b2cef011090d03eedd2a5b81c56d315f6fde4370ba85f2cc98c3c463</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC1850566/pdf/$$EPDF$$P50$$Gpubmedcentral$$H</linktopdf><linktohtml>$$Uhttps://www.sciencedirect.com/science/article/pii/S0002944010617117$$EHTML$$P50$$Gelsevier$$H</linktohtml><link.rule.ids>230,314,723,776,780,881,3537,27901,27902,53766,53768,65534</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=14081866$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/11485898$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Bridge, Julia A.</creatorcontrib><creatorcontrib>Kanamori, Masahiko</creatorcontrib><creatorcontrib>Ma, Zhigui</creatorcontrib><creatorcontrib>Pickering, Diane</creatorcontrib><creatorcontrib>Hill, D. Ashley</creatorcontrib><creatorcontrib>Lydiatt, William</creatorcontrib><creatorcontrib>Lui, Man Yee</creatorcontrib><creatorcontrib>Colleoni, Gisele W.B.</creatorcontrib><creatorcontrib>Antonescu, Cristina R.</creatorcontrib><creatorcontrib>Ladanyi, Marc</creatorcontrib><creatorcontrib>Morris, Stephan W.</creatorcontrib><title>Fusion of the ALK Gene to the Clathrin Heavy Chain Gene, CLTC, in Inflammatory Myofibroblastic Tumor</title><title>The American journal of pathology</title><addtitle>Am J Pathol</addtitle><description>Inflammatory myofibroblastic tumor (IMT) is a rare, but distinctive mesenchymal neoplasm composed of fascicles of bland myofibroblasts admixed with a prominent inflammatory component. Genetic studies of IMTs have demonstrated chromosomal abnormalities of 2p23 and rearrangement of the anaplastic lymphoma kinase (
ALK) gene locus. In a subset of IMTs, the ALK C-terminal kinase domain is fused with a tropomyosin N-terminal coiled-coil domain. In the current study, fusion of
ALK with the clathrin heavy chain (
CTLC) gene localized to 17q23 was detected in two cases of IMT. One of these cases exhibited a 2;17 translocation in addition to other karyotypic anomalies [46,XX,t(2;17)(p23;q23),add(16)(q24)].</description><subject>Adult</subject><subject>Amino Acid Sequence</subject><subject>Base Sequence</subject><subject>Biological and medical sciences</subject><subject>Child, Preschool</subject><subject>Chromosome Mapping</subject><subject>Chromosomes, Human, Pair 2</subject><subject>Clathrin - genetics</subject><subject>Dermatology</subject><subject>Female</subject><subject>Gene Rearrangement</subject><subject>Granuloma, Plasma Cell - genetics</subject><subject>Granuloma, Plasma Cell - pathology</subject><subject>Head and Neck Neoplasms - genetics</subject><subject>Head and Neck Neoplasms - pathology</subject><subject>Humans</subject><subject>In Situ Hybridization, Fluorescence</subject><subject>Karyotyping</subject><subject>Male</subject><subject>Medical sciences</subject><subject>Oncogene Proteins, Fusion - genetics</subject><subject>Pelvic Neoplasms - genetics</subject><subject>Pelvic Neoplasms - pathology</subject><subject>Protein-Tyrosine Kinases - genetics</subject><subject>Receptor Protein-Tyrosine Kinases - genetics</subject><subject>Reverse Transcriptase Polymerase Chain Reaction</subject><subject>Short Communications</subject><subject>Tumors of the skin and soft tissue. 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Ashley</au><au>Lydiatt, William</au><au>Lui, Man Yee</au><au>Colleoni, Gisele W.B.</au><au>Antonescu, Cristina R.</au><au>Ladanyi, Marc</au><au>Morris, Stephan W.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Fusion of the ALK Gene to the Clathrin Heavy Chain Gene, CLTC, in Inflammatory Myofibroblastic Tumor</atitle><jtitle>The American journal of pathology</jtitle><addtitle>Am J Pathol</addtitle><date>2001-08-01</date><risdate>2001</risdate><volume>159</volume><issue>2</issue><spage>411</spage><epage>415</epage><pages>411-415</pages><issn>0002-9440</issn><eissn>1525-2191</eissn><coden>AJPAA4</coden><abstract>Inflammatory myofibroblastic tumor (IMT) is a rare, but distinctive mesenchymal neoplasm composed of fascicles of bland myofibroblasts admixed with a prominent inflammatory component. Genetic studies of IMTs have demonstrated chromosomal abnormalities of 2p23 and rearrangement of the anaplastic lymphoma kinase (
ALK) gene locus. In a subset of IMTs, the ALK C-terminal kinase domain is fused with a tropomyosin N-terminal coiled-coil domain. In the current study, fusion of
ALK with the clathrin heavy chain (
CTLC) gene localized to 17q23 was detected in two cases of IMT. One of these cases exhibited a 2;17 translocation in addition to other karyotypic anomalies [46,XX,t(2;17)(p23;q23),add(16)(q24)].</abstract><cop>Bethesda, MD</cop><pub>Elsevier Inc</pub><pmid>11485898</pmid><doi>10.1016/S0002-9440(10)61711-7</doi><tpages>5</tpages><oa>free_for_read</oa></addata></record> |
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| ispartof | The American journal of pathology, 2001-08, Vol.159 (2), p.411-415 |
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| source | MEDLINE; Elsevier ScienceDirect Journals Complete; Elektronische Zeitschriftenbibliothek - Frei zugängliche E-Journals; PubMed Central |
| subjects | Adult Amino Acid Sequence Base Sequence Biological and medical sciences Child, Preschool Chromosome Mapping Chromosomes, Human, Pair 2 Clathrin - genetics Dermatology Female Gene Rearrangement Granuloma, Plasma Cell - genetics Granuloma, Plasma Cell - pathology Head and Neck Neoplasms - genetics Head and Neck Neoplasms - pathology Humans In Situ Hybridization, Fluorescence Karyotyping Male Medical sciences Oncogene Proteins, Fusion - genetics Pelvic Neoplasms - genetics Pelvic Neoplasms - pathology Protein-Tyrosine Kinases - genetics Receptor Protein-Tyrosine Kinases - genetics Reverse Transcriptase Polymerase Chain Reaction Short Communications Tumors of the skin and soft tissue. Premalignant lesions |
| title | Fusion of the ALK Gene to the Clathrin Heavy Chain Gene, CLTC, in Inflammatory Myofibroblastic Tumor |
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