Diabetes insipidus

[...]excessive persistent fluid intake is called dipsogenic diabetes insipidus, or more commonly primary polydipsia. Cranial diabetes insipidus Familial Autosomal dominant DIDMOAD syndrome Cerebral malformations In association with septo-optic dysplasia Laurence-Moon-Beidl syndrome Acquired Trauma (neurosurgery, head injury) Tumours (such as craniopharyngioma, germinoma, optic glioma) Idiopathic Hypoxic/ischaemic brain damage Lymphocytic neurohypophysitis Granuloma (tuberculosis, sarcoid, histiocytosis) Infections (congenital cytomegalovirus and toxoplamosis, encephalitis, meningitis) Vascular (aneurysm, malformations) Nephrogenic diabetes insipidus Familial X linked recessive inheritance (V2 receptor gene defect) Autosomal recessive inheritance (aquaporin 2 gene defect) Acquired Osmotic diuresis (diabetes mellitus) Metabolic (hypercalcaemia, hypokalaemia) Chronic renal disease Drugs (lithium, demeclocycline) Postobstructive uropathy Solute washout from renal medulla Primary polydipsia Compulsive or habitual In association with psychological disturbance Drugs (lithium, carbamazepine) Hypothalamic lesion DIDMOAD, crainia diabetes insipidus (DI), diabetes mellitus (DM), optic atrophy (OA, and deafness (D).