Heat stress‐induced protection of endothelial function against ischaemic injury is abolished by ATP‐sensitive potassium channel blockade in the isolated rat heart

The protection conferred by heat stress (HS) against myocardial ischaemia‐reperfusion injury, in terms of mechanical function preservation and infarct size reduction, is well documented and mechanisms underlying these effects have been extensively explored. However, the effect of HS on coronary circ...

Ausführliche Beschreibung