The role of nitric oxide, adrenergic activation and kinin‐degradation in blood pressure homeostasis following an acute kinin‐induced hypotension

1 Nitric oxide (NO) has been suggested as the mediator of the vascular response to bradykinin. In the present study, we found that NO did not mediate the hypotensive response to bradykinin. In addition, the significance of kininase II in terminating a kinin‐induced hypotension and the role of the ad...

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Veröffentlicht in:	British journal of pharmacology 1994-12, Vol.113 (4), p.1567-1573
Hauptverfasser:	BJørnstad‐Østensen, Anniken, Berg, Torill
Format:	Artikel
Sprache:	eng
Schlagworte:	Angiotensin-Converting Enzyme Inhibitors - pharmacology Animals Arginine - analogs & derivatives Arginine - pharmacology Arterial hypertension. Arterial hypotension Biological and medical sciences Blood and lymphatic vessels Blood Pressure - physiology blood pressure regulation Bradykinin Bradykinin - pharmacology Cardiology. Vascular system converting enzyme inhibitor EDRF Experimental diseases Heart Rate - drug effects Homeostasis - physiology Hypotension - chemically induced Hypotension - physiopathology kallikrein Kallikreins - pharmacology kininase II Kinins - metabolism Male Medical sciences Nephrectomy NG-Nitroarginine Methyl Ester Nitric Oxide - antagonists & inhibitors Nitric Oxide - physiology NO‐synthase antagonist Phentolamine - pharmacology Phenylephrine - pharmacology Rats Rats, Wistar sympathetic nervous system Sympathetic Nervous System - physiology
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