Control of mitochondrial ATP synthesis in the heart
A number of recent reviews have dealt with the control of oxidative phosphorylation in the heart. In general, however, they have concentrated on the means employed by the cell to modulate the mitochondrial electron transfer chain; the ATP synthase was considered as essentially responding passively t...
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| Veröffentlicht in: | Biochemical journal 1991-12, Vol.280 (3), p.561-573 |
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| container_end_page | 573 |
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| container_title | Biochemical journal |
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| creator | HARRIS, D. A DAS, A. M |
| description | A number of recent reviews have dealt with the control of oxidative phosphorylation in the heart. In general, however, they have concentrated on the means employed by the cell to modulate the mitochondrial electron transfer chain; the ATP synthase was considered as essentially responding passively to changes in these other events. It is the aim of this Review to redress the balance and focus attention on the ATP synthase within heart mitochondria, and on its regulation. |
| doi_str_mv | 10.1042/bj2800561 |
| format | Article |
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A ; DAS, A. M</creator><creatorcontrib>HARRIS, D. A ; DAS, A. M</creatorcontrib><description>A number of recent reviews have dealt with the control of oxidative phosphorylation in the heart. In general, however, they have concentrated on the means employed by the cell to modulate the mitochondrial electron transfer chain; the ATP synthase was considered as essentially responding passively to changes in these other events. It is the aim of this Review to redress the balance and focus attention on the ATP synthase within heart mitochondria, and on its regulation.</description><identifier>ISSN: 0264-6021</identifier><identifier>EISSN: 1470-8728</identifier><identifier>DOI: 10.1042/bj2800561</identifier><identifier>PMID: 1837214</identifier><language>eng</language><publisher>Colchester: Portland Press</publisher><subject>Animals ; ATP ; ATPase Inhibitory Protein ; Biological and medical sciences ; Calcium - metabolism ; Cell physiology ; Down-Regulation ; Fundamental and applied biological sciences. Psychology ; heart ; mechanisms ; mitochondria ; Mitochondria, Heart - metabolism ; Molecular and cellular biology ; Proteins - metabolism ; Proton-Translocating ATPases - antagonists & inhibitors ; Proton-Translocating ATPases - metabolism ; regulation ; reviews ; Secretion. 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A</creatorcontrib><creatorcontrib>DAS, A. M</creatorcontrib><title>Control of mitochondrial ATP synthesis in the heart</title><title>Biochemical journal</title><addtitle>Biochem J</addtitle><description>A number of recent reviews have dealt with the control of oxidative phosphorylation in the heart. In general, however, they have concentrated on the means employed by the cell to modulate the mitochondrial electron transfer chain; the ATP synthase was considered as essentially responding passively to changes in these other events. It is the aim of this Review to redress the balance and focus attention on the ATP synthase within heart mitochondria, and on its regulation.</description><subject>Animals</subject><subject>ATP</subject><subject>ATPase Inhibitory Protein</subject><subject>Biological and medical sciences</subject><subject>Calcium - metabolism</subject><subject>Cell physiology</subject><subject>Down-Regulation</subject><subject>Fundamental and applied biological sciences. Psychology</subject><subject>heart</subject><subject>mechanisms</subject><subject>mitochondria</subject><subject>Mitochondria, Heart - metabolism</subject><subject>Molecular and cellular biology</subject><subject>Proteins - metabolism</subject><subject>Proton-Translocating ATPases - antagonists & inhibitors</subject><subject>Proton-Translocating ATPases - metabolism</subject><subject>regulation</subject><subject>reviews</subject><subject>Secretion. Exocytosis</subject><subject>synthesis</subject><subject>Up-Regulation</subject><issn>0264-6021</issn><issn>1470-8728</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1991</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNqFkUtLw0AUhQdRaq0u_AFCFiK4iN55ZB4boRRfUNBF98MkmZgpSabOpEL_vSktVVeu7oHzcTjcg9AlhjsMjNznSyIBMo6P0BgzAakURB6jMRDOUg4En6KzGJcAmAGDERphSQXBbIzozHd98E3iq6R1vS9q35XBmSaZLt6TuOn62kYXE9clg0pqa0J_jk4q00R7sb8TtHh6XMxe0vnb8-tsOk-LjPI-FVbmylBmOKWWiVIAJ6TMpSgs5qQUxlaUA5NCcFwpLGTJFZOcMFBAc0on6GEXu1rnrS0LOxQ1jV4F15qw0d44_dfpXK0__JfGmAJT24CbfUDwn2sbe926WNimMZ3166gFyRQjKvsXxJwSRoAP4O0OLIKPMdjq0AaD3i6hD0sM7NXv-j_k7vWDf733TSxMUwXTFS4eMKZUhgf2G2QTjnY</recordid><startdate>19911215</startdate><enddate>19911215</enddate><creator>HARRIS, D. A</creator><creator>DAS, A. M</creator><general>Portland Press</general><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>8FD</scope><scope>FR3</scope><scope>M7Z</scope><scope>P64</scope><scope>7X8</scope><scope>5PM</scope></search><sort><creationdate>19911215</creationdate><title>Control of mitochondrial ATP synthesis in the heart</title><author>HARRIS, D. A ; DAS, A. M</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c536t-7e8b9a34a633e47d70622db87ce162d7aef360487761f9178d69486240903b33</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1991</creationdate><topic>Animals</topic><topic>ATP</topic><topic>ATPase Inhibitory Protein</topic><topic>Biological and medical sciences</topic><topic>Calcium - metabolism</topic><topic>Cell physiology</topic><topic>Down-Regulation</topic><topic>Fundamental and applied biological sciences. Psychology</topic><topic>heart</topic><topic>mechanisms</topic><topic>mitochondria</topic><topic>Mitochondria, Heart - metabolism</topic><topic>Molecular and cellular biology</topic><topic>Proteins - metabolism</topic><topic>Proton-Translocating ATPases - antagonists & inhibitors</topic><topic>Proton-Translocating ATPases - metabolism</topic><topic>regulation</topic><topic>reviews</topic><topic>Secretion. Exocytosis</topic><topic>synthesis</topic><topic>Up-Regulation</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>HARRIS, D. A</creatorcontrib><creatorcontrib>DAS, A. M</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>Technology Research Database</collection><collection>Engineering Research Database</collection><collection>Biochemistry Abstracts 1</collection><collection>Biotechnology and BioEngineering Abstracts</collection><collection>MEDLINE - Academic</collection><collection>PubMed Central (Full Participant titles)</collection><jtitle>Biochemical journal</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>HARRIS, D. A</au><au>DAS, A. M</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Control of mitochondrial ATP synthesis in the heart</atitle><jtitle>Biochemical journal</jtitle><addtitle>Biochem J</addtitle><date>1991-12-15</date><risdate>1991</risdate><volume>280</volume><issue>3</issue><spage>561</spage><epage>573</epage><pages>561-573</pages><issn>0264-6021</issn><eissn>1470-8728</eissn><abstract>A number of recent reviews have dealt with the control of oxidative phosphorylation in the heart. In general, however, they have concentrated on the means employed by the cell to modulate the mitochondrial electron transfer chain; the ATP synthase was considered as essentially responding passively to changes in these other events. It is the aim of this Review to redress the balance and focus attention on the ATP synthase within heart mitochondria, and on its regulation.</abstract><cop>Colchester</cop><pub>Portland Press</pub><pmid>1837214</pmid><doi>10.1042/bj2800561</doi><tpages>13</tpages><oa>free_for_read</oa></addata></record> |
| fulltext | fulltext |
| identifier | ISSN: 0264-6021 |
| ispartof | Biochemical journal, 1991-12, Vol.280 (3), p.561-573 |
| issn | 0264-6021 1470-8728 |
| language | eng |
| recordid | cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_1130493 |
| source | MEDLINE; PubMed Central; Alma/SFX Local Collection; EZB Electronic Journals Library |
| subjects | Animals ATP ATPase Inhibitory Protein Biological and medical sciences Calcium - metabolism Cell physiology Down-Regulation Fundamental and applied biological sciences. Psychology heart mechanisms mitochondria Mitochondria, Heart - metabolism Molecular and cellular biology Proteins - metabolism Proton-Translocating ATPases - antagonists & inhibitors Proton-Translocating ATPases - metabolism regulation reviews Secretion. Exocytosis synthesis Up-Regulation |
| title | Control of mitochondrial ATP synthesis in the heart |
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