IL-4 blockade alters the tumor microenvironment and augments the response to cancer immunotherapy in a mouse model

Recent findings show that immune cells constitute a large fraction of the tumor microenvironment and that they modulate tumor progression. Clinical data indicate that chronic inflammation is present at tumor sites and that IL-4, in particular, is upregulated. Thus, we tested whether IL-4 neutralizat...

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Veröffentlicht in:Cancer Immunology, Immunotherapy Immunotherapy, 2017-11, Vol.66 (11), p.1485-1496
Hauptverfasser: Ito, Shuku-ei, Shirota, Hidekazu, Kasahara, Yuki, Saijo, Ken, Ishioka, Chikashi
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container_end_page 1496
container_issue 11
container_start_page 1485
container_title Cancer Immunology, Immunotherapy
container_volume 66
creator Ito, Shuku-ei
Shirota, Hidekazu
Kasahara, Yuki
Saijo, Ken
Ishioka, Chikashi
description Recent findings show that immune cells constitute a large fraction of the tumor microenvironment and that they modulate tumor progression. Clinical data indicate that chronic inflammation is present at tumor sites and that IL-4, in particular, is upregulated. Thus, we tested whether IL-4 neutralization would affect tumor immunity. Current results demonstrate that the administration of a neutralizing antibody against IL-4 enhances anti-tumor immunity and delays tumor progression. IL-4 blockade also alters inflammation in the tumor microenvironment, reducing the generation of both immunosuppressive M2 macrophages and myeloid-derived suppressor cells, and enhancing tumor-specific cytotoxic T lymphocytes. In addition, IL-4 blockade improves the response to anti-OX40 Ab or CpG oligodeoxynucleotide immunotherapies. These findings suggest that IL-4 affects anti-tumor immunity and constitutes an attractive therapeutic target to reduce immune suppression in the tumor microenvironment, thus enhancing the efficacy of cancer therapy.
doi_str_mv 10.1007/s00262-017-2043-6
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source MEDLINE; SpringerLink Journals; PubMed Central
subjects Animals
Antibodies, Neutralizing - immunology
Antibodies, Neutralizing - pharmacology
Cancer
Cancer immunotherapy
Cancer Research
Cell Line, Tumor
CpG islands
Cytotoxicity
Gene Expression Regulation, Neoplastic - drug effects
Gene Expression Regulation, Neoplastic - immunology
Immunology
Immunosuppression
Immunotherapy
Immunotherapy - methods
Interleukin 4
Interleukin-4 - antagonists & inhibitors
Interleukin-4 - genetics
Interleukin-4 - immunology
Lymphocytes
Lymphocytes T
Macrophages
Macrophages - classification
Macrophages - drug effects
Macrophages - immunology
Medicine
Medicine & Public Health
Mice, Inbred BALB C
Neoplasms, Experimental - immunology
Neoplasms, Experimental - pathology
Neoplasms, Experimental - therapy
Neutralization
Oligodeoxyribonucleotides - immunology
Oligodeoxyribonucleotides - pharmacology
Oncology
Original
Original Article
Receptors, OX40 - antagonists & inhibitors
Receptors, OX40 - immunology
Reverse Transcriptase Polymerase Chain Reaction
Suppressor cells
T-Lymphocytes, Cytotoxic - drug effects
T-Lymphocytes, Cytotoxic - immunology
Time Factors
Treatment Outcome
Tumor Burden - drug effects
Tumor Burden - genetics
Tumor Burden - immunology
Tumor Microenvironment - drug effects
Tumor Microenvironment - immunology
Tumor necrosis factor
title IL-4 blockade alters the tumor microenvironment and augments the response to cancer immunotherapy in a mouse model
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