Augmentation of Endothelial S1PR1 Attenuates Postviral Pulmonary Fibrosis

Respiratory viral infections are frequent causes of acute respiratory distress syndrome (ARDS), a disabling condition with a mortality of up to 46%. The pulmonary endothelium plays an important role in the development of ARDS as well as the pathogenesis of pulmonary fibrosis; however, the therapeuti...

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Veröffentlicht in:	American journal of respiratory cell and molecular biology 2024-02, Vol.70 (2), p.119-128
Hauptverfasser:	Brazee, Patricia L, Cartier, Andreane, Kuo, Andrew, Haring, Alexis M, Nguyen, Trong, Hariri, Lida P, Griffith, Jason W, Hla, Timothy, Medoff, Benjamin D, Knipe, Rachel S
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Cells Endothelial cells Endothelium - metabolism Fibrosis Gene expression Humans Inflammation Influenza A Internalization Lung diseases Mice Original Research Orthomyxoviridae Infections Paroxetine Pulmonary Fibrosis Respiratory Distress Syndrome Sphingosine-1-Phosphate Receptors - metabolism Viral infections
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