FLT3 inhibitors upregulate CXCR4 and E-selectin ligands via ERK suppression in AML cells and CXCR4/E-selectin inhibition enhances anti-leukemia efficacy of FLT3-targeted therapy in AML
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| Veröffentlicht in: | Leukemia 2023-06, Vol.37 (6), p.1379-1383 |
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| creator | Jia, Yannan Zhang, Weiguo Basyal, Mahesh Chang, Kyung Hee Ostermann, Lauren Burks, Jared K. Ly, Charlie Mu-Mosley, Hong Zhang, Qi Han, Xin Fogler, William E. Magnani, John L. Lesegretain, Arnaud Zal, Anna A. Zal, Tomasz Andreeff, Michael |
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| doi_str_mv | 10.1038/s41375-023-01897-x |
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AML</title><title>Leukemia</title><addtitle>Leukemia</addtitle><addtitle>Leukemia</addtitle><subject>631/67/1059/2326</subject><subject>692/308/575</subject><subject>96</subject><subject>96/100</subject><subject>96/106</subject><subject>96/2</subject><subject>96/21</subject><subject>96/31</subject><subject>96/35</subject><subject>96/44</subject><subject>96/63</subject><subject>96/95</subject><subject>Acute myeloid leukemia</subject><subject>Cancer Research</subject><subject>Cancer therapies</subject><subject>Cell adhesion & migration</subject><subject>Chemokines</subject><subject>Critical Care Medicine</subject><subject>CXCR4 protein</subject><subject>Cytokines</subject><subject>Drug dosages</subject><subject>E-Selectin</subject><subject>fms-Like Tyrosine Kinase 3</subject><subject>Hematology</subject><subject>Humans</subject><subject>Hypotheses</subject><subject>Hypoxia</subject><subject>Intensive</subject><subject>Internal 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| ispartof | Leukemia, 2023-06, Vol.37 (6), p.1379-1383 |
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| subjects | 631/67/1059/2326 692/308/575 96 96/100 96/106 96/2 96/21 96/31 96/35 96/44 96/63 96/95 Acute myeloid leukemia Cancer Research Cancer therapies Cell adhesion & migration Chemokines Critical Care Medicine CXCR4 protein Cytokines Drug dosages E-Selectin fms-Like Tyrosine Kinase 3 Hematology Humans Hypotheses Hypoxia Intensive Internal Medicine Letter Leukemia Leukemia, Myeloid, Acute - drug therapy Ligands MAP Kinase Signaling System Medical prognosis Medicine Medicine & Public Health Mutation Oncology Patients Protein Kinase Inhibitors - pharmacology Receptors, CXCR4 - genetics Signal Transduction |
| title | FLT3 inhibitors upregulate CXCR4 and E-selectin ligands via ERK suppression in AML cells and CXCR4/E-selectin inhibition enhances anti-leukemia efficacy of FLT3-targeted therapy in AML |
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