MDCT-findings in patients with non-occlusive mesenteric ischemia (NOMI): influence of vasoconstrictor agents
Objectives To evaluate the influence of vasoconstrictor agents (VCAs) on signs of vasoconstriction and bowel ischemia on MDCT detected in patients with non-occlusive mesenteric ischemia (NOMI). Methods This 8-year single-center retrospective study consecutively included all patients with histopathol...
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creator | Topolsky, Antoine Pantet, Olivier Liaudet, Lucas Sempoux, Christine Denys, Alban Knebel, Jean-François Schmidt, Sabine |
description | Objectives
To evaluate the influence of vasoconstrictor agents (VCAs) on signs of vasoconstriction and bowel ischemia on MDCT detected in patients with non-occlusive mesenteric ischemia (NOMI).
Methods
This 8-year single-center retrospective study consecutively included all patients with histopathologically proven NOMI who underwent MDCT ≤ 48 h prior to surgical bowel resection. Two blinded radiologists jointly reviewed each examination for signs of bowel ischemia, abdominal organ infarct, mesenteric vessel size and regularity, and ancillary vascular findings. VCA administration (length and dosage), clinical and biochemical data, risk factors, and outcomes were retrieved from patients’ medical records. Subgroup comparisons were performed.
Results
Ninety patients were included (59 males, mean age 65 years); 40 (44.4%) had received VCAs before MDCT. Overall mortality was 32% (
n
= 29), with no significant difference between the two groups. In patients treated with VCAs, the calibre of the superior mesenteric artery (SMA) was smaller (
p
= 0.032), and vasoconstriction of its branches tended to be more important (
p
= 0.096) than in patients not treated with VCAs. The presence and extent of bowel ischemia did not significantly correlate with VCA administration, but abdominal organ infarcts tended to be more frequent (
p
= 0.005) and involved more organs (
p
= 0.088). The VCA group had lower mean arterial pressure (
p
= 0.006) and lower hemoglobin levels (
p
|
doi_str_mv | 10.1007/s00330-023-09415-4 |
format | Article |
fullrecord | <record><control><sourceid>proquest_pubme</sourceid><recordid>TN_cdi_pubmedcentral_primary_oai_pubmedcentral_nih_gov_10121529</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><sourcerecordid>2804144886</sourcerecordid><originalsourceid>FETCH-LOGICAL-c475t-e9abfa311754f298b07c91f87f7733366720b7d07c789786aac1c3115ec882a73</originalsourceid><addsrcrecordid>eNp9kctuFDEQRS0EIiHwAyyQJTbJwlB-dNtmE6EJj0gJ2YS15fHYM4667cHuHsTf4zAhBBasXPI9datKF6GXFN5QAPm2AnAOBBgnoAXtiHiEDqngjFBQ4vGD-gA9q_UGADQV8ik64H2vWafFIRouzxbXJMS0imldcUx4a6fo01Tx9zhtcMqJZOeGucadx6OvTfIlOhyr2_gxWnz85ery_ORdaw3D7JPzOAe8szW7nOrU0CkXbNe3ls_Rk2CH6l_cvUfo68cP14vP5OLq0_ni_QVxQnYT8doug-WUyk4EptUSpNM0KBmk5LztLhks5ar9SqWl6q111DW8804pZiU_Qqd73-28HP3KtdnFDmZb4mjLD5NtNH8rKW7MOu8MBcpox3RzOL5zKPnb7OtkxnawHwabfJ6rYbLXXHKlaUNf_4Pe5Lmkdp9hCgQVQqm-UWxPuZJrLT7cb0PB3KZp9mmalqb5laYRrenVwzvuW37H1wC-B2qT0tqXP7P_Y_sTlXyrCw</addsrcrecordid><sourcetype>Open Access Repository</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>2804144886</pqid></control><display><type>article</type><title>MDCT-findings in patients with non-occlusive mesenteric ischemia (NOMI): influence of vasoconstrictor agents</title><source>MEDLINE</source><source>SpringerNature Journals</source><creator>Topolsky, Antoine ; Pantet, Olivier ; Liaudet, Lucas ; Sempoux, Christine ; Denys, Alban ; Knebel, Jean-François ; Schmidt, Sabine</creator><creatorcontrib>Topolsky, Antoine ; Pantet, Olivier ; Liaudet, Lucas ; Sempoux, Christine ; Denys, Alban ; Knebel, Jean-François ; Schmidt, Sabine</creatorcontrib><description>Objectives
To evaluate the influence of vasoconstrictor agents (VCAs) on signs of vasoconstriction and bowel ischemia on MDCT detected in patients with non-occlusive mesenteric ischemia (NOMI).
Methods
This 8-year single-center retrospective study consecutively included all patients with histopathologically proven NOMI who underwent MDCT ≤ 48 h prior to surgical bowel resection. Two blinded radiologists jointly reviewed each examination for signs of bowel ischemia, abdominal organ infarct, mesenteric vessel size and regularity, and ancillary vascular findings. VCA administration (length and dosage), clinical and biochemical data, risk factors, and outcomes were retrieved from patients’ medical records. Subgroup comparisons were performed.
Results
Ninety patients were included (59 males, mean age 65 years); 40 (44.4%) had received VCAs before MDCT. Overall mortality was 32% (
n
= 29), with no significant difference between the two groups. In patients treated with VCAs, the calibre of the superior mesenteric artery (SMA) was smaller (
p
= 0.032), and vasoconstriction of its branches tended to be more important (
p
= 0.096) than in patients not treated with VCAs. The presence and extent of bowel ischemia did not significantly correlate with VCA administration, but abdominal organ infarcts tended to be more frequent (
p
= 0.005) and involved more organs (
p
= 0.088). The VCA group had lower mean arterial pressure (
p
= 0.006) and lower hemoglobin levels (
p
< 0.001). Several biomarkers of organ failure and inflammation, differed significantly with VCA use, proving worse clinical condition.
Conclusions
MDCT demonstrates more severe SMA vasoconstriction and tends to show increased abdominal organ infarcts after VCA administration in NOMI patients compared to NOMI patients not treated with VCAs.
Key Points
• In critically ill patients with NOMI, MDCT demonstrates VCA support via increased vasoconstriction of the main SMA and its branches.
• VCA administration in NOMI patients tends to contribute to the development of organ infarcts, as shown on MDCT.
• An important degree of vasoconstriction in NOMI patients may indicate insufficient resuscitation and, thus, help clinicians in further patient management.</description><identifier>ISSN: 1432-1084</identifier><identifier>ISSN: 0938-7994</identifier><identifier>EISSN: 1432-1084</identifier><identifier>DOI: 10.1007/s00330-023-09415-4</identifier><identifier>PMID: 36692594</identifier><language>eng</language><publisher>Berlin/Heidelberg: Springer Berlin Heidelberg</publisher><subject>Abdomen ; Aged ; Biomarkers ; Blood pressure ; Blood vessels ; Diagnostic Radiology ; Gastrointestinal ; Hemoglobin ; Humans ; Imaging ; Infarction ; Internal Medicine ; Interventional Radiology ; Intestine ; Ischemia ; Ischemia - diagnostic imaging ; Male ; Medical records ; Medicine ; Medicine & Public Health ; Mesenteric Ischemia - diagnostic imaging ; Neuroradiology ; Patients ; Radiology ; Retrospective Studies ; Risk factors ; Subgroups ; Tomography, X-Ray Computed ; Ultrasound ; Vasoconstriction ; Vasoconstrictor Agents - pharmacology ; Vasoconstrictor Agents - therapeutic use ; Vasoconstrictors</subject><ispartof>European radiology, 2023-05, Vol.33 (5), p.3627-3637</ispartof><rights>The Author(s) 2023</rights><rights>2023. The Author(s).</rights><rights>The Author(s) 2023. This work is published under http://creativecommons.org/licenses/by/4.0/ (the “License”). Notwithstanding the ProQuest Terms and Conditions, you may use this content in accordance with the terms of the License.</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c475t-e9abfa311754f298b07c91f87f7733366720b7d07c789786aac1c3115ec882a73</citedby><cites>FETCH-LOGICAL-c475t-e9abfa311754f298b07c91f87f7733366720b7d07c789786aac1c3115ec882a73</cites><orcidid>0000-0003-2631-3032</orcidid></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://link.springer.com/content/pdf/10.1007/s00330-023-09415-4$$EPDF$$P50$$Gspringer$$Hfree_for_read</linktopdf><linktohtml>$$Uhttps://link.springer.com/10.1007/s00330-023-09415-4$$EHTML$$P50$$Gspringer$$Hfree_for_read</linktohtml><link.rule.ids>230,314,780,784,885,27924,27925,41488,42557,51319</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/36692594$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Topolsky, Antoine</creatorcontrib><creatorcontrib>Pantet, Olivier</creatorcontrib><creatorcontrib>Liaudet, Lucas</creatorcontrib><creatorcontrib>Sempoux, Christine</creatorcontrib><creatorcontrib>Denys, Alban</creatorcontrib><creatorcontrib>Knebel, Jean-François</creatorcontrib><creatorcontrib>Schmidt, Sabine</creatorcontrib><title>MDCT-findings in patients with non-occlusive mesenteric ischemia (NOMI): influence of vasoconstrictor agents</title><title>European radiology</title><addtitle>Eur Radiol</addtitle><addtitle>Eur Radiol</addtitle><description>Objectives
To evaluate the influence of vasoconstrictor agents (VCAs) on signs of vasoconstriction and bowel ischemia on MDCT detected in patients with non-occlusive mesenteric ischemia (NOMI).
Methods
This 8-year single-center retrospective study consecutively included all patients with histopathologically proven NOMI who underwent MDCT ≤ 48 h prior to surgical bowel resection. Two blinded radiologists jointly reviewed each examination for signs of bowel ischemia, abdominal organ infarct, mesenteric vessel size and regularity, and ancillary vascular findings. VCA administration (length and dosage), clinical and biochemical data, risk factors, and outcomes were retrieved from patients’ medical records. Subgroup comparisons were performed.
Results
Ninety patients were included (59 males, mean age 65 years); 40 (44.4%) had received VCAs before MDCT. Overall mortality was 32% (
n
= 29), with no significant difference between the two groups. In patients treated with VCAs, the calibre of the superior mesenteric artery (SMA) was smaller (
p
= 0.032), and vasoconstriction of its branches tended to be more important (
p
= 0.096) than in patients not treated with VCAs. The presence and extent of bowel ischemia did not significantly correlate with VCA administration, but abdominal organ infarcts tended to be more frequent (
p
= 0.005) and involved more organs (
p
= 0.088). The VCA group had lower mean arterial pressure (
p
= 0.006) and lower hemoglobin levels (
p
< 0.001). Several biomarkers of organ failure and inflammation, differed significantly with VCA use, proving worse clinical condition.
Conclusions
MDCT demonstrates more severe SMA vasoconstriction and tends to show increased abdominal organ infarcts after VCA administration in NOMI patients compared to NOMI patients not treated with VCAs.
Key Points
• In critically ill patients with NOMI, MDCT demonstrates VCA support via increased vasoconstriction of the main SMA and its branches.
• VCA administration in NOMI patients tends to contribute to the development of organ infarcts, as shown on MDCT.
• An important degree of vasoconstriction in NOMI patients may indicate insufficient resuscitation and, thus, help clinicians in further patient management.</description><subject>Abdomen</subject><subject>Aged</subject><subject>Biomarkers</subject><subject>Blood pressure</subject><subject>Blood vessels</subject><subject>Diagnostic Radiology</subject><subject>Gastrointestinal</subject><subject>Hemoglobin</subject><subject>Humans</subject><subject>Imaging</subject><subject>Infarction</subject><subject>Internal Medicine</subject><subject>Interventional Radiology</subject><subject>Intestine</subject><subject>Ischemia</subject><subject>Ischemia - diagnostic imaging</subject><subject>Male</subject><subject>Medical records</subject><subject>Medicine</subject><subject>Medicine & Public Health</subject><subject>Mesenteric Ischemia - diagnostic imaging</subject><subject>Neuroradiology</subject><subject>Patients</subject><subject>Radiology</subject><subject>Retrospective Studies</subject><subject>Risk factors</subject><subject>Subgroups</subject><subject>Tomography, X-Ray Computed</subject><subject>Ultrasound</subject><subject>Vasoconstriction</subject><subject>Vasoconstrictor Agents - pharmacology</subject><subject>Vasoconstrictor Agents - therapeutic use</subject><subject>Vasoconstrictors</subject><issn>1432-1084</issn><issn>0938-7994</issn><issn>1432-1084</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2023</creationdate><recordtype>article</recordtype><sourceid>C6C</sourceid><sourceid>EIF</sourceid><sourceid>ABUWG</sourceid><sourceid>AFKRA</sourceid><sourceid>AZQEC</sourceid><sourceid>BENPR</sourceid><sourceid>CCPQU</sourceid><sourceid>DWQXO</sourceid><sourceid>GNUQQ</sourceid><recordid>eNp9kctuFDEQRS0EIiHwAyyQJTbJwlB-dNtmE6EJj0gJ2YS15fHYM4667cHuHsTf4zAhBBasXPI9datKF6GXFN5QAPm2AnAOBBgnoAXtiHiEDqngjFBQ4vGD-gA9q_UGADQV8ik64H2vWafFIRouzxbXJMS0imldcUx4a6fo01Tx9zhtcMqJZOeGucadx6OvTfIlOhyr2_gxWnz85ery_ORdaw3D7JPzOAe8szW7nOrU0CkXbNe3ls_Rk2CH6l_cvUfo68cP14vP5OLq0_ni_QVxQnYT8doug-WUyk4EptUSpNM0KBmk5LztLhks5ar9SqWl6q111DW8804pZiU_Qqd73-28HP3KtdnFDmZb4mjLD5NtNH8rKW7MOu8MBcpox3RzOL5zKPnb7OtkxnawHwabfJ6rYbLXXHKlaUNf_4Pe5Lmkdp9hCgQVQqm-UWxPuZJrLT7cb0PB3KZp9mmalqb5laYRrenVwzvuW37H1wC-B2qT0tqXP7P_Y_sTlXyrCw</recordid><startdate>20230501</startdate><enddate>20230501</enddate><creator>Topolsky, Antoine</creator><creator>Pantet, Olivier</creator><creator>Liaudet, Lucas</creator><creator>Sempoux, Christine</creator><creator>Denys, Alban</creator><creator>Knebel, Jean-François</creator><creator>Schmidt, Sabine</creator><general>Springer Berlin Heidelberg</general><general>Springer Nature B.V</general><scope>C6C</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>3V.</scope><scope>7QO</scope><scope>7RV</scope><scope>7X7</scope><scope>7XB</scope><scope>88E</scope><scope>8AO</scope><scope>8FD</scope><scope>8FE</scope><scope>8FG</scope><scope>8FH</scope><scope>8FI</scope><scope>8FJ</scope><scope>8FK</scope><scope>ABUWG</scope><scope>AFKRA</scope><scope>ARAPS</scope><scope>AZQEC</scope><scope>BBNVY</scope><scope>BENPR</scope><scope>BGLVJ</scope><scope>BHPHI</scope><scope>CCPQU</scope><scope>DWQXO</scope><scope>FR3</scope><scope>FYUFA</scope><scope>GHDGH</scope><scope>GNUQQ</scope><scope>HCIFZ</scope><scope>K9.</scope><scope>KB0</scope><scope>LK8</scope><scope>M0S</scope><scope>M1P</scope><scope>M7P</scope><scope>NAPCQ</scope><scope>P5Z</scope><scope>P62</scope><scope>P64</scope><scope>PQEST</scope><scope>PQQKQ</scope><scope>PQUKI</scope><scope>PRINS</scope><scope>7X8</scope><scope>5PM</scope><orcidid>https://orcid.org/0000-0003-2631-3032</orcidid></search><sort><creationdate>20230501</creationdate><title>MDCT-findings in patients with non-occlusive mesenteric ischemia (NOMI): influence of vasoconstrictor agents</title><author>Topolsky, Antoine ; Pantet, Olivier ; Liaudet, Lucas ; Sempoux, Christine ; Denys, Alban ; Knebel, Jean-François ; Schmidt, Sabine</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c475t-e9abfa311754f298b07c91f87f7733366720b7d07c789786aac1c3115ec882a73</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2023</creationdate><topic>Abdomen</topic><topic>Aged</topic><topic>Biomarkers</topic><topic>Blood pressure</topic><topic>Blood vessels</topic><topic>Diagnostic Radiology</topic><topic>Gastrointestinal</topic><topic>Hemoglobin</topic><topic>Humans</topic><topic>Imaging</topic><topic>Infarction</topic><topic>Internal Medicine</topic><topic>Interventional Radiology</topic><topic>Intestine</topic><topic>Ischemia</topic><topic>Ischemia - diagnostic imaging</topic><topic>Male</topic><topic>Medical records</topic><topic>Medicine</topic><topic>Medicine & Public Health</topic><topic>Mesenteric Ischemia - diagnostic imaging</topic><topic>Neuroradiology</topic><topic>Patients</topic><topic>Radiology</topic><topic>Retrospective Studies</topic><topic>Risk factors</topic><topic>Subgroups</topic><topic>Tomography, X-Ray Computed</topic><topic>Ultrasound</topic><topic>Vasoconstriction</topic><topic>Vasoconstrictor Agents - pharmacology</topic><topic>Vasoconstrictor Agents - therapeutic use</topic><topic>Vasoconstrictors</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Topolsky, Antoine</creatorcontrib><creatorcontrib>Pantet, Olivier</creatorcontrib><creatorcontrib>Liaudet, Lucas</creatorcontrib><creatorcontrib>Sempoux, Christine</creatorcontrib><creatorcontrib>Denys, Alban</creatorcontrib><creatorcontrib>Knebel, Jean-François</creatorcontrib><creatorcontrib>Schmidt, Sabine</creatorcontrib><collection>Springer Nature OA Free Journals</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>ProQuest Central (Corporate)</collection><collection>Biotechnology Research Abstracts</collection><collection>Proquest Nursing & Allied Health Source</collection><collection>Health & Medical Collection</collection><collection>ProQuest Central (purchase pre-March 2016)</collection><collection>Medical Database (Alumni Edition)</collection><collection>ProQuest Pharma Collection</collection><collection>Technology Research Database</collection><collection>ProQuest SciTech Collection</collection><collection>ProQuest Technology Collection</collection><collection>ProQuest Natural Science Collection</collection><collection>Hospital Premium Collection</collection><collection>Hospital Premium Collection (Alumni Edition)</collection><collection>ProQuest Central (Alumni) (purchase pre-March 2016)</collection><collection>ProQuest Central (Alumni Edition)</collection><collection>ProQuest Central UK/Ireland</collection><collection>Advanced Technologies & Aerospace Collection</collection><collection>ProQuest Central Essentials</collection><collection>Biological Science Collection</collection><collection>ProQuest Central</collection><collection>Technology Collection</collection><collection>Natural Science Collection</collection><collection>ProQuest One Community College</collection><collection>ProQuest Central Korea</collection><collection>Engineering Research Database</collection><collection>Health Research Premium Collection</collection><collection>Health Research Premium Collection (Alumni)</collection><collection>ProQuest Central Student</collection><collection>SciTech Premium Collection</collection><collection>ProQuest Health & Medical Complete (Alumni)</collection><collection>Nursing & Allied Health Database (Alumni Edition)</collection><collection>ProQuest Biological Science Collection</collection><collection>Health & Medical Collection (Alumni Edition)</collection><collection>Medical Database</collection><collection>Biological Science Database</collection><collection>Nursing & Allied Health Premium</collection><collection>Advanced Technologies & Aerospace Database</collection><collection>ProQuest Advanced Technologies & Aerospace Collection</collection><collection>Biotechnology and BioEngineering Abstracts</collection><collection>ProQuest One Academic Eastern Edition (DO NOT USE)</collection><collection>ProQuest One Academic</collection><collection>ProQuest One Academic UKI Edition</collection><collection>ProQuest Central China</collection><collection>MEDLINE - Academic</collection><collection>PubMed Central (Full Participant titles)</collection><jtitle>European radiology</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Topolsky, Antoine</au><au>Pantet, Olivier</au><au>Liaudet, Lucas</au><au>Sempoux, Christine</au><au>Denys, Alban</au><au>Knebel, Jean-François</au><au>Schmidt, Sabine</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>MDCT-findings in patients with non-occlusive mesenteric ischemia (NOMI): influence of vasoconstrictor agents</atitle><jtitle>European radiology</jtitle><stitle>Eur Radiol</stitle><addtitle>Eur Radiol</addtitle><date>2023-05-01</date><risdate>2023</risdate><volume>33</volume><issue>5</issue><spage>3627</spage><epage>3637</epage><pages>3627-3637</pages><issn>1432-1084</issn><issn>0938-7994</issn><eissn>1432-1084</eissn><abstract>Objectives
To evaluate the influence of vasoconstrictor agents (VCAs) on signs of vasoconstriction and bowel ischemia on MDCT detected in patients with non-occlusive mesenteric ischemia (NOMI).
Methods
This 8-year single-center retrospective study consecutively included all patients with histopathologically proven NOMI who underwent MDCT ≤ 48 h prior to surgical bowel resection. Two blinded radiologists jointly reviewed each examination for signs of bowel ischemia, abdominal organ infarct, mesenteric vessel size and regularity, and ancillary vascular findings. VCA administration (length and dosage), clinical and biochemical data, risk factors, and outcomes were retrieved from patients’ medical records. Subgroup comparisons were performed.
Results
Ninety patients were included (59 males, mean age 65 years); 40 (44.4%) had received VCAs before MDCT. Overall mortality was 32% (
n
= 29), with no significant difference between the two groups. In patients treated with VCAs, the calibre of the superior mesenteric artery (SMA) was smaller (
p
= 0.032), and vasoconstriction of its branches tended to be more important (
p
= 0.096) than in patients not treated with VCAs. The presence and extent of bowel ischemia did not significantly correlate with VCA administration, but abdominal organ infarcts tended to be more frequent (
p
= 0.005) and involved more organs (
p
= 0.088). The VCA group had lower mean arterial pressure (
p
= 0.006) and lower hemoglobin levels (
p
< 0.001). Several biomarkers of organ failure and inflammation, differed significantly with VCA use, proving worse clinical condition.
Conclusions
MDCT demonstrates more severe SMA vasoconstriction and tends to show increased abdominal organ infarcts after VCA administration in NOMI patients compared to NOMI patients not treated with VCAs.
Key Points
• In critically ill patients with NOMI, MDCT demonstrates VCA support via increased vasoconstriction of the main SMA and its branches.
• VCA administration in NOMI patients tends to contribute to the development of organ infarcts, as shown on MDCT.
• An important degree of vasoconstriction in NOMI patients may indicate insufficient resuscitation and, thus, help clinicians in further patient management.</abstract><cop>Berlin/Heidelberg</cop><pub>Springer Berlin Heidelberg</pub><pmid>36692594</pmid><doi>10.1007/s00330-023-09415-4</doi><tpages>11</tpages><orcidid>https://orcid.org/0000-0003-2631-3032</orcidid><oa>free_for_read</oa></addata></record> |
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ispartof | European radiology, 2023-05, Vol.33 (5), p.3627-3637 |
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language | eng |
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source | MEDLINE; SpringerNature Journals |
subjects | Abdomen Aged Biomarkers Blood pressure Blood vessels Diagnostic Radiology Gastrointestinal Hemoglobin Humans Imaging Infarction Internal Medicine Interventional Radiology Intestine Ischemia Ischemia - diagnostic imaging Male Medical records Medicine Medicine & Public Health Mesenteric Ischemia - diagnostic imaging Neuroradiology Patients Radiology Retrospective Studies Risk factors Subgroups Tomography, X-Ray Computed Ultrasound Vasoconstriction Vasoconstrictor Agents - pharmacology Vasoconstrictor Agents - therapeutic use Vasoconstrictors |
title | MDCT-findings in patients with non-occlusive mesenteric ischemia (NOMI): influence of vasoconstrictor agents |
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