Dietary energy restriction abolishes development of prolactin-producing pituitary tumors in Fischer 344 rats treated with 17beta-estradiol

Reduction in energy consumption is known to inhibit development of a variety of spontaneous, carcinogen-induced, and hormone-dependent cancers, but the mechanism or mechanisms by which this occurs remain unknown. We hypothesize that energy consumption may modulate development of estrogen-dependent n...

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Veröffentlicht in:	Molecular carcinogenesis 1998-10, Vol.23 (2), p.86
Hauptverfasser:	Spady, T J, Lemus-Wilson, A M, Pennington, K L, Blackwood, D J, Paschall, T M, Birt, D F, McComb, R D, Shull, J D
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Apoptosis Cell Survival Diet Energy Intake Estradiol - administration & dosage Estradiol - blood Female Pituitary Neoplasms - metabolism Pituitary Neoplasms - pathology Pituitary Neoplasms - prevention & control Prolactin - biosynthesis Rats Rats, Inbred F344
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