Activation of Nicotinic Receptor-Induced Postsynaptic Responses to Luteinizing Hormone-Releasing Hormone in Bullfrog Sympathetic Ganglia via a Na+-Dependent Mechanism

Nicotine at very low doses (5-30 nM) induced large amounts of luteinizing hormone-releasing hormone (LHRH) release, which was monitored as slow membrane depolarizations in the ganglionic neurons of bullfrog sympathetic ganglia. A nicotinic antagonist, d-tubocurarine chloride, completely and reversib...

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Veröffentlicht in:	Proceedings of the National Academy of Sciences - PNAS 1998-10, Vol.95 (21), p.12689-12694
Hauptverfasser:	Cao, Ying-Jun, Peng, Yan-Yi
Format:	Artikel
Sprache:	eng
Schlagworte:	Action potentials Animals B lymphocytes Biological Sciences Calcium - physiology Cholinergic receptors Depolarization Frogs Ganglia Ganglia, Sympathetic - physiology Gonadotropin-Releasing Hormone - physiology Hormones Ion Channel Gating Luteinization Nerves Neurology Neurons Neuropeptides Nicotine Nicotine - pharmacology Nicotinic Agonists - pharmacology Rana catesbeiana Receptors, Nicotinic - physiology Sodium Channels - physiology Sympathetic ganglia Synapses - physiology
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