Prenatal Exposure to Cocaine Reduces Dopaminergic D1-Mediated Motor Function but Spares the Enhancement of Learning by Amphetamine in Rabbits

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Veröffentlicht in:Annals of the New York Academy of Sciences 1998-06, Vol.846 (1), p.375-378
Hauptverfasser: SIMANSKY, K. J., BAKER, G., KACHELRIES, W. J., HOOD, H., ROMANO, A. G., HARVEY, J. A.
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container_title Annals of the New York Academy of Sciences
container_volume 846
creator SIMANSKY, K. J.
BAKER, G.
KACHELRIES, W. J.
HOOD, H.
ROMANO, A. G.
HARVEY, J. A.
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doi_str_mv 10.1111/j.1749-6632.1998.tb09759.x
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source MEDLINE; Wiley Online Library Journals Frontfile Complete
subjects Acoustic Stimulation
Animals
Association Learning - drug effects
Benzazepines - pharmacology
Blinking - drug effects
Blinking - physiology
Catalepsy - etiology
Catalepsy - physiopathology
Cocaine - toxicity
Conditioning, Operant - drug effects
Dextroamphetamine - pharmacology
Female
Locomotion - drug effects
Male
Motor Activity - drug effects
Motor Activity - physiology
Nictitating Membrane
Pregnancy
Prenatal Exposure Delayed Effects
Rabbits
Receptors, Dopamine D1 - physiology
Sex Characteristics
Stereotyped Behavior - drug effects
title Prenatal Exposure to Cocaine Reduces Dopaminergic D1-Mediated Motor Function but Spares the Enhancement of Learning by Amphetamine in Rabbits
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