The Escherichia coli K-12 gntP gene allows E. coli F-18 to occupy a distinct nutritional niche in the streptomycin-treated mouse large intestine
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Veröffentlicht in: | Infection and Immunity 1996-09, Vol.64 (9), p.3497-3503 |
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creator | Sweeney, N J Klemm, P McCormick, B A Moller-Nielsen, E Utley, M Schembri, M A Laux, D C Cohen, P S |
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</description><identifier>ISSN: 0019-9567</identifier><identifier>EISSN: 1098-5522</identifier><identifier>DOI: 10.1128/iai.64.9.3497-3503.1996</identifier><identifier>PMID: 8751890</identifier><language>eng</language><publisher>United States: American Society for Microbiology</publisher><subject>Animals ; Anti-Bacterial Agents - pharmacology ; Escherichia coli ; Escherichia coli - genetics ; Escherichia coli - pathogenicity ; Escherichia coli Proteins ; Genes, Bacterial ; Intestine, Large - microbiology ; Male ; Membrane Transport Proteins - genetics ; Membrane Transport Proteins - physiology ; Mice ; Mice, Inbred Strains ; Streptomycin - pharmacology</subject><ispartof>Infection and Immunity, 1996-09, Vol.64 (9), p.3497-3503</ispartof><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c522t-cdd8dc60f6b61d5a480aa772e603e97cd0b97f26ffa069c43d7a1d1775911a223</citedby></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC174254/pdf/$$EPDF$$P50$$Gpubmedcentral$$H</linktopdf><linktohtml>$$Uhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC174254/$$EHTML$$P50$$Gpubmedcentral$$H</linktohtml><link.rule.ids>230,314,727,780,784,885,3188,3189,27924,27925,53791,53793</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/8751890$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Sweeney, N J</creatorcontrib><creatorcontrib>Klemm, P</creatorcontrib><creatorcontrib>McCormick, B A</creatorcontrib><creatorcontrib>Moller-Nielsen, E</creatorcontrib><creatorcontrib>Utley, M</creatorcontrib><creatorcontrib>Schembri, M A</creatorcontrib><creatorcontrib>Laux, D C</creatorcontrib><creatorcontrib>Cohen, P S</creatorcontrib><title>The Escherichia coli K-12 gntP gene allows E. coli F-18 to occupy a distinct nutritional niche in the streptomycin-treated mouse large intestine</title><title>Infection and Immunity</title><addtitle>Infect Immun</addtitle><description>Services
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</description><subject>Animals</subject><subject>Anti-Bacterial Agents - pharmacology</subject><subject>Escherichia coli</subject><subject>Escherichia coli - genetics</subject><subject>Escherichia coli - pathogenicity</subject><subject>Escherichia coli Proteins</subject><subject>Genes, Bacterial</subject><subject>Intestine, Large - microbiology</subject><subject>Male</subject><subject>Membrane Transport Proteins - genetics</subject><subject>Membrane Transport Proteins - physiology</subject><subject>Mice</subject><subject>Mice, Inbred Strains</subject><subject>Streptomycin - pharmacology</subject><issn>0019-9567</issn><issn>1098-5522</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1996</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNqFkctu1DAUhi0EKtPCIyDMhl2C7cS3BQtUTSmiEizK2vLYzsQosQfboZq34JFxNKOKrlgdW_9F5-gD4C1GLcZEfPDat6xvZdv1kjcdRV2LpWTPwAYjKRpKCXkONghh2UjK-EtwmfPP-u37XlyAC8EpFhJtwJ_70cFtNqNL3oxeQxMnD782mMB9KN_h3gUH9TTFhwy37Um9abCAJcJozHI4Qg2tz8UHU2BYSvLFx6AnGGqfgz7AUkcuyR1KnI_Gh6a-dXEWznHJDk467VdfcWuJewVeDHrK7vV5XoEfN9v769vm7tvnL9ef7hpTTyuNsVZYw9DAdgxbqnuBtOacOIY6J7mxaCf5QNgwaMSk6TvLNbaYcyox1oR0V-Djqfew7GZnjQsl6Ukdkp91OqqovXqqBD-qffytMO8J7Wv-_Tmf4q-l7q5mn42bJh1cvUtxQZgUtPuvEVPWSc5FNfKT0aSYc3LD4zIYqRW6qtAV65VUK3S1Qlcr9Jp88-8tj7kz5aq_O-mj348PPjml8_y0rfsL_8a27Q</recordid><startdate>19960901</startdate><enddate>19960901</enddate><creator>Sweeney, N J</creator><creator>Klemm, P</creator><creator>McCormick, B A</creator><creator>Moller-Nielsen, E</creator><creator>Utley, M</creator><creator>Schembri, M A</creator><creator>Laux, D C</creator><creator>Cohen, P S</creator><general>American Society for Microbiology</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7QL</scope><scope>8FD</scope><scope>C1K</scope><scope>FR3</scope><scope>P64</scope><scope>RC3</scope><scope>7X8</scope><scope>5PM</scope></search><sort><creationdate>19960901</creationdate><title>The Escherichia coli K-12 gntP gene allows E. coli F-18 to occupy a distinct nutritional niche in the streptomycin-treated mouse large intestine</title><author>Sweeney, N J ; 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</abstract><cop>United States</cop><pub>American Society for Microbiology</pub><pmid>8751890</pmid><doi>10.1128/iai.64.9.3497-3503.1996</doi><tpages>7</tpages><oa>free_for_read</oa></addata></record> |
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source | MEDLINE; American Society for Microbiology Journals; EZB-FREE-00999 freely available EZB journals; PubMed Central |
subjects | Animals Anti-Bacterial Agents - pharmacology Escherichia coli Escherichia coli - genetics Escherichia coli - pathogenicity Escherichia coli Proteins Genes, Bacterial Intestine, Large - microbiology Male Membrane Transport Proteins - genetics Membrane Transport Proteins - physiology Mice Mice, Inbred Strains Streptomycin - pharmacology |
title | The Escherichia coli K-12 gntP gene allows E. coli F-18 to occupy a distinct nutritional niche in the streptomycin-treated mouse large intestine |
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