Hepatic P38 Activation Modulates Systemic Metabolism Through Fgf21-Mediated Interorgan Communication
The mechanisms underlying the pathogenesis of steatosis and insulin resistance in nonalcoholic fatty liver disease remain elusive. Increased phosphorylation of hepatic p38 has long been noticed in fatty liver; however, whether the activation of hepatic p38 is a cause or consequence of liver steatosi...
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Veröffentlicht in: | Diabetes (New York, N.Y.) N.Y.), 2021-10 |
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Hauptverfasser: | , , , , , , , , , , , , , , , |
Format: | Artikel |
Sprache: | eng |
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